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384 related items for PubMed ID: 9416842

  • 21. Identification and characterization of genes whose expressions are altered in rat 6 fibroblasts transformed by mutant p53(val135).
    Yam JW, Zheng JY, Hsiao WL.
    Biochem Biophys Res Commun; 1999 Dec 20; 266(2):472-80. PubMed ID: 10600527
    [Abstract] [Full Text] [Related]

  • 22. Dose-dependent oncogene-induced senescence in vivo and its evasion during mammary tumorigenesis.
    Sarkisian CJ, Keister BA, Stairs DB, Boxer RB, Moody SE, Chodosh LA.
    Nat Cell Biol; 2007 May 20; 9(5):493-505. PubMed ID: 17450133
    [Abstract] [Full Text] [Related]

  • 23. Substrate-induced polarisation of cultured epitheliocytes and fibroblasts: non-reactivity of Ras-transformed cells.
    Moizhess TG, Vasiliev JM.
    Cell Biol Int; 2001 May 20; 25(9):931-4. PubMed ID: 11518500
    [Abstract] [Full Text] [Related]

  • 24. Stepwise neoplastic transformation of a telomerase immortalized fibroblast cell line.
    Zongaro S, de Stanchina E, Colombo T, D'Incalci M, Giulotto E, Mondello C.
    Cancer Res; 2005 Dec 15; 65(24):11411-8. PubMed ID: 16357149
    [Abstract] [Full Text] [Related]

  • 25. Activated oncogenes promote and cooperate with chromosomal instability for neoplastic transformation.
    Woo RA, Poon RY.
    Genes Dev; 2004 Jun 01; 18(11):1317-30. PubMed ID: 15175263
    [Abstract] [Full Text] [Related]

  • 26. Ornithine decarboxylase induction in transformation by H-Ras and RhoA.
    Shantz LM, Pegg AE.
    Cancer Res; 1998 Jul 01; 58(13):2748-53. PubMed ID: 9661886
    [Abstract] [Full Text] [Related]

  • 27. Knock-in of mutant K-ras in nontumorigenic human epithelial cells as a new model for studying K-ras mediated transformation.
    Konishi H, Karakas B, Abukhdeir AM, Lauring J, Gustin JP, Garay JP, Konishi Y, Gallmeier E, Bachman KE, Park BH.
    Cancer Res; 2007 Sep 15; 67(18):8460-7. PubMed ID: 17875684
    [Abstract] [Full Text] [Related]

  • 28. Mutant p53 DNA clones from human colon carcinomas cooperate with ras in transforming primary rat cells: a comparison of the "hot spot" mutant phenotypes.
    Hinds PW, Finlay CA, Quartin RS, Baker SJ, Fearon ER, Vogelstein B, Levine AJ.
    Cell Growth Differ; 1990 Dec 15; 1(12):571-80. PubMed ID: 2288874
    [Abstract] [Full Text] [Related]

  • 29. A modest reduction in c-myc expression has minimal effects on cell growth and apoptosis but dramatically reduces susceptibility to Ras and Raf transformation.
    Bazarov AV, Adachi S, Li SF, Mateyak MK, Wei S, Sedivy JM.
    Cancer Res; 2001 Feb 01; 61(3):1178-86. PubMed ID: 11221849
    [Abstract] [Full Text] [Related]

  • 30. [Expression of p53 and ras p21 gene products in malignant transformed V79 cell induced by various organic components of DEPs].
    Lü Y, Ding X, Ye S.
    Wei Sheng Yan Jiu; 1998 Jul 01; 27(4):224-6. PubMed ID: 10682588
    [Abstract] [Full Text] [Related]

  • 31. Murine fibroblasts lacking p21 undergo senescence and are resistant to transformation by oncogenic Ras.
    Pantoja C, Serrano M.
    Oncogene; 1999 Sep 02; 18(35):4974-82. PubMed ID: 10490832
    [Abstract] [Full Text] [Related]

  • 32. Poly(ADP-ribose) polymerase-1 regulates the stability of the wild-type p53 protein.
    Wesierska-Gadek J, Schmid G.
    Cell Mol Biol Lett; 2001 Sep 02; 6(2):117-40. PubMed ID: 11544635
    [Abstract] [Full Text] [Related]

  • 33.
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  • 34. Role of the Ha-ras (RasH) oncogene in mediating progression of the tumor cell phenotype (review).
    Boylan JF, Jackson J, Steiner MR, Shih TY, Duigou GJ, Roszman T, Fisher PB, Zimmer SG.
    Anticancer Res; 1990 Sep 02; 10(3):717-24. PubMed ID: 2195986
    [Abstract] [Full Text] [Related]

  • 35. Malignant transformation of rat embryo fibroblasts by cotransfection with eleven human mutant p53 cDNAs and activated H-ras gene.
    Kikuchi-Yanoshita R, Tanaka K, Muraoka M, Konishi M, Kawashima I, Takamoto S, Hirai H, Miyaki M.
    Oncogene; 1995 Oct 05; 11(7):1339-45. PubMed ID: 7478555
    [Abstract] [Full Text] [Related]

  • 36. Multiple aspects of the phenotype of mammary epithelial cells transformed by expression of activated M-Ras depend on an autocrine mechanism mediated by hepatocyte growth factor/scatter factor.
    Zhang KX, Ward KR, Schrader JW.
    Mol Cancer Res; 2004 Apr 05; 2(4):242-55. PubMed ID: 15140946
    [Abstract] [Full Text] [Related]

  • 37. A dominant negative RAS-specific guanine nucleotide exchange factor reverses neoplastic phenotype in K-ras transformed mouse fibroblasts.
    Bossù P, Vanoni M, Wanke V, Cesaroni MP, Tropea F, Melillo G, Asti C, Porzio S, Ruggiero P, Di Cioccio V, Maurizi G, Ciabini A, Alberghina L.
    Oncogene; 2000 Apr 20; 19(17):2147-54. PubMed ID: 10815806
    [Abstract] [Full Text] [Related]

  • 38. Length control is determined by the pattern of cytoskeleton.
    Kharitonova MA, Vasiliev JM.
    J Cell Sci; 2004 Apr 15; 117(Pt 10):1955-60. PubMed ID: 15039460
    [Abstract] [Full Text] [Related]

  • 39. Escape from premature senescence is not sufficient for oncogenic transformation by Ras.
    Peeper DS, Dannenberg JH, Douma S, te Riele H, Bernards R.
    Nat Cell Biol; 2001 Feb 15; 3(2):198-203. PubMed ID: 11175753
    [Abstract] [Full Text] [Related]

  • 40. While E1A can facilitate epithelial cell transformation by several dominant oncogenes, the C-terminus seems only to regulate rac and cdc42 function, but in both epithelial and fibroblastic cells.
    Fischer RS, Quinlan MP.
    Virology; 2000 Apr 10; 269(2):404-19. PubMed ID: 10753719
    [Abstract] [Full Text] [Related]

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