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Journal Abstract Search

239 related items for PubMed ID: 9439509

  • 1. Are neuronal intranuclear inclusions the common neuropathology of triplet-repeat disorders with polyglutamine-repeat expansions?
    Davies SW, Beardsall K, Turmaine M, DiFiglia M, Aronin N, Bates GP.
    Lancet; 1998 Jan 10; 351(9096):131-3. PubMed ID: 9439509
    [Abstract] [Full Text] [Related]

  • 2. Neuronal distribution of intranuclear inclusions in Huntington's disease with adult onset.
    Gourfinkel-An I, Cancel G, Duyckaerts C, Faucheux B, Hauw JJ, Trottier Y, Brice A, Agid Y, Hirsch EC.
    Neuroreport; 1998 Jun 01; 9(8):1823-6. PubMed ID: 9665608
    [Abstract] [Full Text] [Related]

  • 3. Transgenic mice expressing mutated full-length HD cDNA: a paradigm for locomotor changes and selective neuronal loss in Huntington's disease.
    Reddy PH, Charles V, Williams M, Miller G, Whetsell WO, Tagle DA.
    Philos Trans R Soc Lond B Biol Sci; 1999 Jun 29; 354(1386):1035-45. PubMed ID: 10434303
    [Abstract] [Full Text] [Related]

  • 4. Intranuclear neuronal inclusions in Huntington's disease and dentatorubral and pallidoluysian atrophy: correlation between the density of inclusions and IT15 CAG triplet repeat length.
    Becher MW, Kotzuk JA, Sharp AH, Davies SW, Bates GP, Price DL, Ross CA.
    Neurobiol Dis; 1998 Apr 29; 4(6):387-97. PubMed ID: 9666478
    [Abstract] [Full Text] [Related]

  • 5. Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation.
    Davies SW, Turmaine M, Cozens BA, DiFiglia M, Sharp AH, Ross CA, Scherzinger E, Wanker EE, Mangiarini L, Bates GP.
    Cell; 1997 Aug 08; 90(3):537-48. PubMed ID: 9267033
    [Abstract] [Full Text] [Related]

  • 6. Paradoxical delay in the onset of disease caused by super-long CAG repeat expansions in R6/2 mice.
    Morton AJ, Glynn D, Leavens W, Zheng Z, Faull RL, Skepper JN, Wight JM.
    Neurobiol Dis; 2009 Mar 08; 33(3):331-41. PubMed ID: 19130884
    [Abstract] [Full Text] [Related]

  • 7. Transgenic models of Huntington's disease.
    Sathasivam K, Hobbs C, Mangiarini L, Mahal A, Turmaine M, Doherty P, Davies SW, Bates GP.
    Philos Trans R Soc Lond B Biol Sci; 1999 Jun 29; 354(1386):963-9. PubMed ID: 10434294
    [Abstract] [Full Text] [Related]

  • 8. Polyglutamine expansions cause decreased CRE-mediated transcription and early gene expression changes prior to cell death in an inducible cell model of Huntington's disease.
    Wyttenbach A, Swartz J, Kita H, Thykjaer T, Carmichael J, Bradley J, Brown R, Maxwell M, Schapira A, Orntoft TF, Kato K, Rubinsztein DC.
    Hum Mol Genet; 2001 Aug 15; 10(17):1829-45. PubMed ID: 11532992
    [Abstract] [Full Text] [Related]

  • 9. Properties of polyglutamine expansion in vitro and in a cellular model for Huntington's disease.
    Lunkes A, Trottier Y, Fagart J, Schultz P, Zeder-Lutz G, Moras D, Mandel JL.
    Philos Trans R Soc Lond B Biol Sci; 1999 Jun 29; 354(1386):1013-9. PubMed ID: 10434300
    [Abstract] [Full Text] [Related]

  • 10. Polyglutamines, nuclear inclusions and neurodegeneration.
    Lunkes A, Mandel JL.
    Nat Med; 1997 Nov 29; 3(11):1201-2. PubMed ID: 9359692
    [No Abstract] [Full Text] [Related]

  • 11. Polyglutamine pathogenesis.
    Ross CA, Wood JD, Schilling G, Peters MF, Nucifora FC, Cooper JK, Sharp AH, Margolis RL, Borchelt DR.
    Philos Trans R Soc Lond B Biol Sci; 1999 Jun 29; 354(1386):1005-11. PubMed ID: 10434299
    [Abstract] [Full Text] [Related]

  • 12. Huntington's disease intranuclear inclusions contain truncated, ubiquitinated huntingtin protein.
    Sieradzan KA, Mechan AO, Jones L, Wanker EE, Nukina N, Mann DM.
    Exp Neurol; 1999 Mar 29; 156(1):92-9. PubMed ID: 10192780
    [Abstract] [Full Text] [Related]

  • 13. Intranuclear neuronal inclusions: a common pathogenic mechanism for glutamine-repeat neurodegenerative diseases?
    Ross CA.
    Neuron; 1997 Dec 29; 19(6):1147-50. PubMed ID: 9427237
    [No Abstract] [Full Text] [Related]

  • 14. Bioenergetics in Huntington's disease.
    Grünewald T, Beal MF.
    Ann N Y Acad Sci; 1999 Dec 29; 893():203-13. PubMed ID: 10672239
    [Abstract] [Full Text] [Related]

  • 15. Biochemical, ultrastructural, and reversibility studies on huntingtin filaments isolated from mouse and human brain.
    Díaz-Hernández M, Moreno-Herrero F, Gómez-Ramos P, Morán MA, Ferrer I, Baró AM, Avila J, Hernández F, Lucas JJ.
    J Neurosci; 2004 Oct 20; 24(42):9361-71. PubMed ID: 15496672
    [Abstract] [Full Text] [Related]

  • 16. Intranuclear inclusions and neuritic aggregates in transgenic mice expressing a mutant N-terminal fragment of huntingtin.
    Schilling G, Becher MW, Sharp AH, Jinnah HA, Duan K, Kotzuk JA, Slunt HH, Ratovitski T, Cooper JK, Jenkins NA, Copeland NG, Price DL, Ross CA, Borchelt DR.
    Hum Mol Genet; 1999 Mar 20; 8(3):397-407. PubMed ID: 9949199
    [Abstract] [Full Text] [Related]

  • 17. Formation of polyglutamine inclusions in non-CNS tissue.
    Sathasivam K, Hobbs C, Turmaine M, Mangiarini L, Mahal A, Bertaux F, Wanker EE, Doherty P, Davies SW, Bates GP.
    Hum Mol Genet; 1999 May 20; 8(5):813-22. PubMed ID: 10196370
    [Abstract] [Full Text] [Related]

  • 18. Clinico-pathological rescue of a model mouse of Huntington's disease by siRNA.
    Wang YL, Liu W, Wada E, Murata M, Wada K, Kanazawa I.
    Neurosci Res; 2005 Nov 20; 53(3):241-9. PubMed ID: 16095740
    [Abstract] [Full Text] [Related]

  • 19. Adenovirus vector-based in vitro neuronal cell model for Huntington's disease with human disease-like differential aggregation and degeneration.
    Dong X, Zong S, Witting A, Lindenberg KS, Kochanek S, Huang B.
    J Gene Med; 2012 Jul 20; 14(7):468-81. PubMed ID: 22700462
    [Abstract] [Full Text] [Related]

  • 20. Formation of polyglutamine inclusions in a wide range of non-CNS tissues in the HdhQ150 knock-in mouse model of Huntington's disease.
    Moffitt H, McPhail GD, Woodman B, Hobbs C, Bates GP.
    PLoS One; 2009 Nov 30; 4(11):e8025. PubMed ID: 19956633
    [Abstract] [Full Text] [Related]

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