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Journal Abstract Search

271 related items for PubMed ID: 9458856

  • 1. Preconditioning in rat hearts is independent of mitochondrial F1F0 ATPase inhibition.
    Green DW, Murray HN, Sleph PG, Wang FL, Baird AJ, Rogers WL, Grover GJ.
    Am J Physiol; 1998 Jan; 274(1):H90-7. PubMed ID: 9458856
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  • 2. Mechanisms of ischemic preconditioning in rat myocardium. Roles of adenosine, cellular energy state, and mitochondrial F1F0-ATPase.
    Vuorinen K, Ylitalo K, Peuhkurinen K, Raatikainen P, Ala-Rämi A, Hassinen IE.
    Circulation; 1995 Jun 01; 91(11):2810-8. PubMed ID: 7758188
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  • 3. Myocardial ischemic preconditioning and mitochondrial F1F0-ATPase activity.
    Bosetti F, Yu G, Zucchi R, Ronca-Testoni S, Solaini G.
    Mol Cell Biochem; 2000 Dec 01; 215(1-2):31-7. PubMed ID: 11204453
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  • 5. Excessive ATP hydrolysis in ischemic myocardium by mitochondrial F1F0-ATPase: effect of selective pharmacological inhibition of mitochondrial ATPase hydrolase activity.
    Grover GJ, Atwal KS, Sleph PG, Wang FL, Monshizadegan H, Monticello T, Green DW.
    Am J Physiol Heart Circ Physiol; 2004 Oct 01; 287(4):H1747-55. PubMed ID: 15371268
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  • 8. Ischemic preconditioning with opening of mitochondrial adenosine triphosphate-sensitive potassium channels or Na/H exchange inhibition: which is the best protective strategy for heart transplants?
    Kevelaitis E, Oubénaissa A, Mouas C, Peynet J, Menasché P.
    J Thorac Cardiovasc Surg; 2001 Jan 01; 121(1):155-62. PubMed ID: 11135172
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  • 9. Reversible ischemic inhibition of F(1)F(0)-ATPase in rat and human myocardium.
    Ylitalo K, Ala-Rämi A, Vuorinen K, Peuhkurinen K, Lepojärvi M, Kaukoranta P, Kiviluoma K, Hassinen I.
    Biochim Biophys Acta; 2001 Apr 02; 1504(2-3):329-39. PubMed ID: 11245796
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  • 15. Transgenic expression of Bcl-2 modulates energy metabolism, prevents cytosolic acidification during ischemia, and reduces ischemia/reperfusion injury.
    Imahashi K, Schneider MD, Steenbergen C, Murphy E.
    Circ Res; 2004 Oct 01; 95(7):734-41. PubMed ID: 15345651
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  • 16. IF1 function in situ in uncoupler-challenged ischemic rabbit, rat, and pigeon hearts.
    Rouslin W, Broge CW.
    J Biol Chem; 1996 Sep 27; 271(39):23638-41. PubMed ID: 8798581
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  • 18. Preconditioning accelerates contracture and ATP depletion in blood-perfused rat hearts.
    Kolocassides KG, Galiñanes M, Hearse DJ.
    Am J Physiol; 1995 Oct 27; 269(4 Pt 2):H1415-20. PubMed ID: 7485575
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  • 19. Dystrophin is a possible end-target of ischemic preconditioning against cardiomyocyte oncosis during the early phase of reperfusion.
    Kyoi S, Otani H, Hamano A, Matsuhisa S, Akita Y, Fujiwara H, Hattori R, Imamura H, Kamihata H, Iwasaka T.
    Cardiovasc Res; 2006 May 01; 70(2):354-63. PubMed ID: 16466703
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