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678 related items for PubMed ID: 9467563

  • 1. Hyperfunctioning thyroid nodules in toxic multinodular goiter share activating thyrotropin receptor mutations with solitary toxic adenoma.
    Tonacchera M, Chiovato L, Pinchera A, Agretti P, Fiore E, Cetani F, Rocchi R, Viacava P, Miccoli P, Vitti P.
    J Clin Endocrinol Metab; 1998 Feb; 83(2):492-8. PubMed ID: 9467563
    [Abstract] [Full Text] [Related]

  • 2. Activating thyrotropin receptor mutations are present in nonadenomatous hyperfunctioning nodules of toxic or autonomous multinodular goiter.
    Tonacchera M, Agretti P, Chiovato L, Rosellini V, Ceccarini G, Perri A, Viacava P, Naccarato AG, Miccoli P, Pinchera A, Vitti P.
    J Clin Endocrinol Metab; 2000 Jun; 85(6):2270-4. PubMed ID: 10852462
    [Abstract] [Full Text] [Related]

  • 3. Constitutively activating TSH receptor mutations as the cause of toxic thyroid adenoma, multinodular toxic goiter and autosomal dominant non autoimmune hyperthyroidism.
    Paschke R.
    Exp Clin Endocrinol Diabetes; 1996 Jun; 104 Suppl 4():129-32. PubMed ID: 8981020
    [Abstract] [Full Text] [Related]

  • 4. Activating thyrotropin receptor mutations in histologically heterogeneous hyperfunctioning nodules of multinodular goiter.
    Tonacchera M, Vitti P, Agretti P, Giulianetti B, Mazzi B, Cavaliere R, Ceccarini G, Fiore E, Viacava P, Naccarato A, Pinchera A, Chiovato L.
    Thyroid; 1998 Jul; 8(7):559-64. PubMed ID: 9709907
    [Abstract] [Full Text] [Related]

  • 5. Functioning and nonfunctioning thyroid adenomas involve different molecular pathogenetic mechanisms.
    Tonacchera M, Vitti P, Agretti P, Ceccarini G, Perri A, Cavaliere R, Mazzi B, Naccarato AG, Viacava P, Miccoli P, Pinchera A, Chiovato L.
    J Clin Endocrinol Metab; 1999 Nov; 84(11):4155-8. PubMed ID: 10566665
    [Abstract] [Full Text] [Related]

  • 6. Non-hyperfunctioning nodules from multinodular goiters: a minor role in pathogenesis for somatic activating mutations in the TSH-receptor and Gsalpha subunit genes.
    Derrien C, Sonnet E, Gicquel I, Le Gall JY, Poirier JY, David V, Maugendre D.
    J Endocrinol Invest; 2001 May; 24(5):321-5. PubMed ID: 11407651
    [Abstract] [Full Text] [Related]

  • 7. [Mutations in the thyrotropin receptor gene in the pathogenesis of toxic thyroid adenomas, toxic goiter nodules and autosomal dominant hyperthyroidism].
    Derwahl M.
    Z Arztl Fortbild Qualitatssich; 1999 Apr; 93 Suppl 1():25-8. PubMed ID: 10355046
    [Abstract] [Full Text] [Related]

  • 8. [Non-autoimmune hyperthyroidism and hyperfunctioning thyroid adenomas caused by activating mutation of the thyrotropin receptor].
    Kosugi S.
    Nihon Rinsho; 2002 Feb; 60(2):291-6. PubMed ID: 11857916
    [Abstract] [Full Text] [Related]

  • 9. Rarity of oncogenic mutations in the thyrotropin receptor of autonomously functioning thyroid nodules in Japan.
    Takeshita A, Nagayama Y, Yokoyama N, Ishikawa N, Ito K, Yamashita T, Obara T, Murakami Y, Kuma K, Takamatsu J.
    J Clin Endocrinol Metab; 1995 Sep; 80(9):2607-11. PubMed ID: 7673402
    [Abstract] [Full Text] [Related]

  • 10. Pathogenesis of toxic thyroid adenomas and nodules: relevance of activating mutations in the TSH-receptor and Gs-alpha gene, the possible role of iodine deficiency and secondary and TSH-independent molecular mechanisms.
    Derwahl M, Manole D, Sobke A, Broecker M.
    Exp Clin Endocrinol Diabetes; 1998 Sep; 106 Suppl 4():S6-9. PubMed ID: 9867188
    [Abstract] [Full Text] [Related]

  • 11. Thyrotropin receptor mutations in hyperfunctioning thyroid adenomas from Brazil.
    Nogueira CR, Kopp P, Arseven OK, Santos CL, Jameson JL, Medeiros-Neto G.
    Thyroid; 1999 Nov; 9(11):1063-8. PubMed ID: 10595453
    [Abstract] [Full Text] [Related]

  • 12. Somatic mutations causing constitutive activity of the thyrotropin receptor are the major cause of hyperfunctioning thyroid adenomas: identification of additional mutations activating both the cyclic adenosine 3',5'-monophosphate and inositol phosphate-Ca2+ cascades.
    Parma J, Van Sande J, Swillens S, Tonacchera M, Dumont J, Vassart G.
    Mol Endocrinol; 1995 Jun; 9(6):725-33. PubMed ID: 8592518
    [Abstract] [Full Text] [Related]

  • 13. Two novel mutations in the sixth transmembrane segment of the thyrotropin receptor gene causing hyperfunctioning thyroid nodules.
    Gozu H, Avsar M, Bircan R, Claus M, Sahin S, Sezgin O, Deyneli O, Paschke R, Cirakoglu B, Akalin S.
    Thyroid; 2005 Apr; 15(4):389-97. PubMed ID: 15876166
    [Abstract] [Full Text] [Related]

  • 14. Diversity and prevalence of somatic mutations in the thyrotropin receptor and Gs alpha genes as a cause of toxic thyroid adenomas.
    Parma J, Duprez L, Van Sande J, Hermans J, Rocmans P, Van Vliet G, Costagliola S, Rodien P, Dumont JE, Vassart G.
    J Clin Endocrinol Metab; 1997 Aug; 82(8):2695-701. PubMed ID: 9253356
    [Abstract] [Full Text] [Related]

  • 15. Autonomously functioning thyroid nodules in a former iodine-deficient area commonly harbor gain-of-function mutations in the thyrotropin signaling pathway.
    Georgopoulos NA, Sykiotis GP, Sgourou A, Papachatzopoulou A, Markou KB, Kyriazopoulou V, Papavassiliou AG, Vagenakis AG.
    Eur J Endocrinol; 2003 Oct; 149(4):287-92. PubMed ID: 14514342
    [Abstract] [Full Text] [Related]

  • 16. Molecular aspects of the pathogenesis of nodular goiters, thyroid nodules and adenomas.
    Derwahl M.
    Exp Clin Endocrinol Diabetes; 1996 Oct; 104 Suppl 4():32-5. PubMed ID: 8980997
    [Abstract] [Full Text] [Related]

  • 17. Hyperfunctioning thyroid adenoma and activating mutations in the TSH receptor gene.
    Polak M.
    Arch Med Res; 1999 Oct; 30(6):510-3. PubMed ID: 10714365
    [Abstract] [Full Text] [Related]

  • 18. Identification and functional characterization of two new somatic mutations causing constitutive activation of the thyrotropin receptor in hyperfunctioning autonomous adenomas of the thyroid.
    Paschke R, Tonacchera M, Van Sande J, Parma J, Vassart G.
    J Clin Endocrinol Metab; 1994 Dec; 79(6):1785-9. PubMed ID: 7989485
    [Abstract] [Full Text] [Related]

  • 19. Identification of constitutively activating somatic thyrotropin receptor mutations in a subset of toxic multinodular goiters.
    Holzapfel HP, Führer D, Wonerow P, Weinland G, Scherbaum WA, Paschke R.
    J Clin Endocrinol Metab; 1997 Dec; 82(12):4229-33. PubMed ID: 9398745
    [Abstract] [Full Text] [Related]

  • 20. Lack of association of nonautoimmune hyperfunctioning thyroid disorders and a germline polymorphism of codon 727 of the human thyrotropin receptor in a European Caucasian population.
    Mühlberg T, Herrmann K, Joba W, Kirchberger M, Heberling HJ, Heufelder AE.
    J Clin Endocrinol Metab; 2000 Aug; 85(8):2640-3. PubMed ID: 10946859
    [Abstract] [Full Text] [Related]

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