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Journal Abstract Search

501 related items for PubMed ID: 9482107

  • 1. Transformation suppressor activity of C3G is independent of its CDC25-homology domain.
    Guerrero C, Fernandez-Medarde A, Rojas JM, Font de Mora J, Esteban LM, Santos E.
    Oncogene; 1998 Feb 05; 16(5):613-24. PubMed ID: 9482107
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  • 5. Rac-1 dependent stimulation of the JNK/SAPK signaling pathway by Vav.
    Crespo P, Bustelo XR, Aaronson DS, Coso OA, Lopez-Barahona M, Barbacid M, Gutkind JS.
    Oncogene; 1996 Aug 01; 13(3):455-60. PubMed ID: 8760286
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  • 8. A role for Ras in v-Crk transformation.
    Greulich H, Hanafusa H.
    Cell Growth Differ; 1996 Nov 01; 7(11):1443-51. PubMed ID: 8930393
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  • 9. Vav cooperates with Ras to transform rodent fibroblasts but is not a Ras GDP/GTP exchange factor.
    Bustelo XR, Suen KL, Leftheris K, Meyers CA, Barbacid M.
    Oncogene; 1994 Aug 01; 9(8):2405-13. PubMed ID: 8036025
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  • 10. Ras-15A protein shares highly similar dominant-negative biological properties with Ras-17N and forms a stable, guanine-nucleotide resistant complex with CDC25 exchange factor.
    Chen SY, Huff SY, Lai CC, Der CJ, Powers S.
    Oncogene; 1994 Sep 01; 9(9):2691-8. PubMed ID: 8058333
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  • 13. Biochemical characterization of C3G: an exchange factor that discriminates between Rap1 and Rap2 and is not inhibited by Rap1A(S17N).
    van den Berghe N, Cool RH, Horn G, Wittinghofer A.
    Oncogene; 1997 Aug 14; 15(7):845-50. PubMed ID: 9266971
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  • 14. Inhibition of oncogene-mediated transformation by ectopic expression of p21Waf1 in NIH3T3 cells.
    Michieli P, Li W, Lorenzi MV, Miki T, Zakut R, Givol D, Pierce JH.
    Oncogene; 1996 Feb 15; 12(4):775-84. PubMed ID: 8632899
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  • 15. Rit, a non-lipid-modified Ras-related protein, transforms NIH3T3 cells without activating the ERK, JNK, p38 MAPK or PI3K/Akt pathways.
    Rusyn EV, Reynolds ER, Shao H, Grana TM, Chan TO, Andres DA, Cox AD.
    Oncogene; 2000 Sep 28; 19(41):4685-94. PubMed ID: 11032018
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  • 16. smg/rap1/Krev-1 p21s inhibit the signal pathway to the c-fos promoter/enhancer from c-Ki-ras p21 but not from c-raf-1 kinase in NIH3T3 cells.
    Sakoda T, Kaibuchi K, Kishi K, Kishida S, Doi K, Hoshino M, Hattori S, Takai Y.
    Oncogene; 1992 Sep 28; 7(9):1705-11. PubMed ID: 1323817
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  • 17. Identification of Rap1 as a target for the Crk SH3 domain-binding guanine nucleotide-releasing factor C3G.
    Gotoh T, Hattori S, Nakamura S, Kitayama H, Noda M, Takai Y, Kaibuchi K, Matsui H, Hatase O, Takahashi H.
    Mol Cell Biol; 1995 Dec 28; 15(12):6746-53. PubMed ID: 8524240
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  • 18. RalGDS-like factor (Rlf) is a novel Ras and Rap 1A-associating protein.
    Wolthuis RM, Bauer B, van 't Veer LJ, de Vries-Smits AM, Cool RH, Spaargaren M, Wittinghofer A, Burgering BM, Bos JL.
    Oncogene; 1996 Jul 18; 13(2):353-62. PubMed ID: 8710374
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  • 19. The non-catalytic domain of ras-GAP inhibits transformation induced by G protein coupled receptors.
    Xu N, McCormick F, Gutkind JS.
    Oncogene; 1994 Feb 18; 9(2):597-601. PubMed ID: 8290270
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  • 20. Isoform-specific insertion near the Grb2-binding domain modulates the intrinsic guanine nucleotide exchange activity of hSos1.
    Rojas JM, Subleski M, Coque JJ, Guerrero C, Saez R, Li BQ, Lopez E, Zarich N, Aroca P, Kamata T, Santos E.
    Oncogene; 1999 Mar 04; 18(9):1651-61. PubMed ID: 10208427
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