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Journal Abstract Search

221 related items for PubMed ID: 9482942

  • 1. Point mutations in segment I-S6 render voltage-gated Na+ channels resistant to batrachotoxin.
    Wang SY, Wang GK.
    Proc Natl Acad Sci U S A; 1998 Mar 03; 95(5):2653-8. PubMed ID: 9482942
    [Abstract] [Full Text] [Related]

  • 2. Residues in Na(+) channel D3-S6 segment modulate both batrachotoxin and local anesthetic affinities.
    Wang SY, Nau C, Wang GK.
    Biophys J; 2000 Sep 03; 79(3):1379-87. PubMed ID: 10969000
    [Abstract] [Full Text] [Related]

  • 3. Batrachotoxin-resistant Na+ channels derived from point mutations in transmembrane segment D4-S6.
    Wang SY, Wang GK.
    Biophys J; 1999 Jun 03; 76(6):3141-9. PubMed ID: 10354438
    [Abstract] [Full Text] [Related]

  • 4. Modification of wild-type and batrachotoxin-resistant muscle mu1 Na+ channels by veratridine.
    Wang GK, Quan C, Seaver M, Wang SY.
    Pflugers Arch; 2000 Apr 03; 439(6):705-13. PubMed ID: 10784344
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  • 5. Point mutations at N434 in D1-S6 of mu1 Na(+) channels modulate binding affinity and stereoselectivity of local anesthetic enantiomers.
    Nau C, Wang SY, Strichartz GR, Wang GK.
    Mol Pharmacol; 1999 Aug 03; 56(2):404-13. PubMed ID: 10419561
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  • 6. Disparate role of Na(+) channel D2-S6 residues in batrachotoxin and local anesthetic action.
    Wang SY, Barile M, Wang GK.
    Mol Pharmacol; 2001 May 03; 59(5):1100-7. PubMed ID: 11306693
    [Abstract] [Full Text] [Related]

  • 7. A phenylalanine residue at segment D3-S6 in Nav1.4 voltage-gated Na(+) channels is critical for pyrethroid action.
    Wang SY, Barile M, Wang GK.
    Mol Pharmacol; 2001 Sep 03; 60(3):620-8. PubMed ID: 11502895
    [Abstract] [Full Text] [Related]

  • 8. Point-mutations related to the loss of batrachotoxin binding abolish the grayanotoxin effect in Na(+) channel isoforms.
    Ishii H, Kinoshita E, Kimura T, Yakehiro M, Yamaoka K, Imoto K, Mori Y, Seyama I.
    Jpn J Physiol; 1999 Oct 03; 49(5):457-61. PubMed ID: 10603430
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  • 13. External pore residue mediates slow inactivation in mu 1 rat skeletal muscle sodium channels.
    Balser JR, Nuss HB, Chiamvimonvat N, Pérez-García MT, Marban E, Tomaselli GF.
    J Physiol; 1996 Jul 15; 494 ( Pt 2)(Pt 2):431-42. PubMed ID: 8842002
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  • 14. Comparison of slow inactivation in human heart and rat skeletal muscle Na+ channel chimaeras.
    O'Reilly JP, Wang SY, Kallen RG, Wang GK.
    J Physiol; 1999 Feb 15; 515 ( Pt 1)(Pt 1):61-73. PubMed ID: 9925878
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  • 16. How batrachotoxin modifies the sodium channel permeation pathway: computer modeling and site-directed mutagenesis.
    Wang SY, Mitchell J, Tikhonov DB, Zhorov BS, Wang GK.
    Mol Pharmacol; 2006 Mar 15; 69(3):788-95. PubMed ID: 16354762
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  • 18. Coupling between fast and slow inactivation revealed by analysis of a point mutation (F1304Q) in mu 1 rat skeletal muscle sodium channels.
    Nuss HB, Balser JR, Orias DW, Lawrence JH, Tomaselli GF, Marban E.
    J Physiol; 1996 Jul 15; 494 ( Pt 2)(Pt 2):411-29. PubMed ID: 8842001
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  • 19. Time-dependent block and resurgent tail currents induced by mouse beta4(154-167) peptide in cardiac Na+ channels.
    Wang GK, Edrich T, Wang SY.
    J Gen Physiol; 2006 Mar 15; 127(3):277-89. PubMed ID: 16505148
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  • 20. A mutation in segment I-S6 alters slow inactivation of sodium channels.
    Wang SY, Wang GK.
    Biophys J; 1997 Apr 15; 72(4):1633-40. PubMed ID: 9083667
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