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Journal Abstract Search

182 related items for PubMed ID: 9490700

  • 1. Impaired Ca2+-induced tyrosine phosphorylation and defective lipid scrambling in erythrocytes from a patient with Scott syndrome: a study using an inhibitor for scramblase that mimics the defect in Scott syndrome.
    Dekkers DW, Comfurius P, Vuist WM, Billheimer JT, Dicker I, Weiss HJ, Zwaal RF, Bevers EM.
    Blood; 1998 Mar 15; 91(6):2133-8. PubMed ID: 9490700
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  • 2. Defective Ca(2+)-induced microvesiculation and deficient expression of procoagulant activity in erythrocytes from a patient with a bleeding disorder: a study of the red blood cells of Scott syndrome.
    Bevers EM, Wiedmer T, Comfurius P, Shattil SJ, Weiss HJ, Zwaal RF, Sims PJ.
    Blood; 1992 Jan 15; 79(2):380-8. PubMed ID: 1730083
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  • 3. Scott syndrome erythrocytes contain a membrane protein capable of mediating Ca2+-dependent transbilayer migration of membrane phospholipids.
    Stout JG, Bassé F, Luhm RA, Weiss HJ, Wiedmer T, Sims PJ.
    J Clin Invest; 1997 May 01; 99(9):2232-8. PubMed ID: 9151796
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  • 7. Ca2+ sensitivity of phospholipid scrambling in human red cell ghosts.
    Woon LA, Holland JW, Kable EP, Roufogalis BD.
    Cell Calcium; 1999 Apr 01; 25(4):313-20. PubMed ID: 10456228
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  • 8. Calcium induces phospholipid redistribution and microvesicle release in human erythrocyte membranes by independent pathways.
    Bucki R, Bachelot-Loza C, Zachowski A, Giraud F, Sulpice JC.
    Biochemistry; 1998 Nov 03; 37(44):15383-91. PubMed ID: 9799499
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  • 9. Scott syndrome, a bleeding disorder caused by defective scrambling of membrane phospholipids.
    Zwaal RF, Comfurius P, Bevers EM.
    Biochim Biophys Acta; 2004 Mar 22; 1636(2-3):119-28. PubMed ID: 15164759
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  • 12. Collagen but not fibrinogen surfaces induce bleb formation, exposure of phosphatidylserine, and procoagulant activity of adherent platelets: evidence for regulation by protein tyrosine kinase-dependent Ca2+ responses.
    Heemskerk JW, Vuist WM, Feijge MA, Reutelingsperger CP, Lindhout T.
    Blood; 1997 Oct 01; 90(7):2615-25. PubMed ID: 9326228
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  • 13. Significance of capacitative Ca2+ entry in the regulation of phosphatidylserine expression at the surface of stimulated cells.
    Martínez MC, Martin S, Toti F, Fressinaud E, Dachary-Prigent J, Meyer D, Freyssinet JM.
    Biochemistry; 1999 Aug 03; 38(31):10092-8. PubMed ID: 10433717
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  • 16. Influence of erythrocyte shape on the rate of Ca2+-induced scrambling of phosphatidylserine.
    Wolfs JL, Comfurius P, Bevers EM, Zwaal RF.
    Mol Membr Biol; 2003 Aug 03; 20(1):83-91. PubMed ID: 12745928
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  • 17. Phospholipid scramblase activation pathways in lymphocytes.
    Williamson P, Christie A, Kohlin T, Schlegel RA, Comfurius P, Harmsma M, Zwaal RF, Bevers EM.
    Biochemistry; 2001 Jul 10; 40(27):8065-72. PubMed ID: 11434775
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  • 18. Regulatory mechanisms of transmembrane phospholipid distributions and pathophysiological implications of transbilayer lipid scrambling.
    Bevers EM, Comfurius P, Dekkers DW, Harmsma M, Zwaal RF.
    Lupus; 1998 Jul 10; 7 Suppl 2():S126-31. PubMed ID: 9814689
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  • 20. A TMEM16F point mutation causes an absence of canine platelet TMEM16F and ineffective activation and death-induced phospholipid scrambling.
    Brooks MB, Catalfamo JL, MacNguyen R, Tim D, Fancher S, McCardle JA.
    J Thromb Haemost; 2015 Dec 10; 13(12):2240-52. PubMed ID: 26414452
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