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215 related items for PubMed ID: 9537261

  • 1. Overexpressed WAF1/Cip1 renders glioblastoma cells resistant to chemotherapy agents 1,3-bis(2-chloroethyl)-1-nitrosourea and cisplatin.
    Ruan S, Okcu MF, Ren JP, Chiao P, Andreeff M, Levin V, Zhang W.
    Cancer Res; 1998 Apr 01; 58(7):1538-43. PubMed ID: 9537261
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  • 2. Attenuation of WAF1/Cip1 expression by an antisense adenovirus expression vector sensitizes glioblastoma cells to apoptosis induced by chemotherapeutic agents 1,3-bis(2-chloroethyl)-1-nitrosourea and cisplatin.
    Ruan S, Okcu MF, Pong RC, Andreeff M, Levin V, Hsieh JT, Zhang W.
    Clin Cancer Res; 1999 Jan 01; 5(1):197-202. PubMed ID: 9918219
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  • 11. Potent iron chelators increase the mRNA levels of the universal cyclin-dependent kinase inhibitor p21(CIP1/WAF1), but paradoxically inhibit its translation: a potential mechanism of cell cycle dysregulation.
    Le NT, Richardson DR.
    Carcinogenesis; 2003 Jun 01; 24(6):1045-58. PubMed ID: 12807743
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  • 13. A dual role of p21waf1/cip1 gene in apoptosis of HEp-2 treated with cisplatin or methotrexate.
    Kraljevic Pavelic S, Cacev T, Kralj M.
    Cancer Gene Ther; 2008 Sep 01; 15(9):576-90. PubMed ID: 18483502
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  • 17. p21 Waf1/Cip1 expression by curcumin in U-87MG human glioma cells: role of early growth response-1 expression.
    Choi BH, Kim CG, Bae YS, Lim Y, Lee YH, Shin SY.
    Cancer Res; 2008 Mar 01; 68(5):1369-77. PubMed ID: 18316600
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  • 18. A new molecular role for iron in regulation of cell cycling and differentiation of HL-60 human leukemia cells: iron is required for transcription of p21(WAF1/CIP1) in cells induced by phorbol myristate acetate.
    Gazitt Y, Reddy SV, Alcantara O, Yang J, Boldt DH.
    J Cell Physiol; 2001 Apr 01; 187(1):124-35. PubMed ID: 11241357
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  • 19. Endogenous p21WAF1/CIP1 status predicts the response of human tumor cells to wild-type p53 and p21WAF1/CIP1 overexpression.
    Kralj M, Husnjak K, Körbler T, Pavelić J.
    Cancer Gene Ther; 2003 Jun 01; 10(6):457-67. PubMed ID: 12768191
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  • 20. Differential sensitivity of malignant glioma cells to methylating and chloroethylating anticancer drugs: p53 determines the switch by regulating xpc, ddb2, and DNA double-strand breaks.
    Batista LF, Roos WP, Christmann M, Menck CF, Kaina B.
    Cancer Res; 2007 Dec 15; 67(24):11886-95. PubMed ID: 18089819
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