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Journal Abstract Search

199 related items for PubMed ID: 9543387

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  • 5. Induction of a complex between rasGAP and a novel 110 kD protein is required for immortalization of primary epithelial cells by the E1A 12S oncoprotein of adenovirus.
    Gopalakrishnan S, Fischer RS, Quinlan MP.
    Oncogene; 1996 Dec 19; 13(12):2659-69. PubMed ID: 9000140
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  • 6. The C terminus of E1A regulates tumor progression and epithelial cell differentiation.
    Fischer RS, Quinlan MP.
    Virology; 1998 Sep 30; 249(2):427-39. PubMed ID: 9791033
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  • 8. The adenovirus E4orf6 protein contributes to malignant transformation by antagonizing E1A-induced accumulation of the tumor suppressor protein p53.
    Nevels M, Spruss T, Wolf H, Dobner T.
    Oncogene; 1999 Jan 07; 18(1):9-17. PubMed ID: 9926915
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  • 9. [Constitutive activity of MAP kinase cascades in REF cells transformed by E1A and cHa-ras oncogenes].
    Svetlikova SB, Abramova MV, Kukushkin AN, Darieva ZA, Pospelova TV, Pospelov VA.
    Tsitologiia; 2001 Jan 07; 43(10):961-8. PubMed ID: 11769129
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  • 10. Specific association of activated MAP kinase kinase kinase (Raf) with the plasma membranes of ras-transformed retinal cells.
    Traverse S, Cohen P, Paterson H, Marshall C, Rapp U, Grand RJ.
    Oncogene; 1993 Nov 07; 8(11):3175-81. PubMed ID: 8414521
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  • 11. An essential role for Rac in Ras transformation.
    Qiu RG, Chen J, Kirn D, McCormick F, Symons M.
    Nature; 1995 Mar 30; 374(6521):457-9. PubMed ID: 7700355
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  • 12. [Rat embryo fibroblasts transformed by complementation with oncogenes E1A+E1B-19 and E1A+cHa-ras differ in the ability to realize the G1/S block in serum free media].
    Brichkina AI, Tararova ND, Aksenov ND, Pospelov VA, Pospelova TV.
    Tsitologiia; 2001 Mar 30; 43(11):1024-30. PubMed ID: 11840777
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  • 14. Efficient nuclear localization of the Ad5 E1A 12S protein is necessary for immortalization but not cotransformation of primary epithelial cells.
    Douglas JL, Quinlan MP.
    Cell Growth Differ; 1994 May 30; 5(5):475-83. PubMed ID: 8049154
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  • 15. Ras-interacting domain of Ral GDP dissociation stimulator like (RGL) reverses v-Ras-induced transformation and Raf-1 activation in NIH3T3 cells.
    Okazaki M, Kishida S, Murai H, Hinoi T, Kikuchi A.
    Cancer Res; 1996 May 15; 56(10):2387-92. PubMed ID: 8625316
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  • 16. Enhanced proliferation, growth factor induction and immortalization by adenovirus E1A 12S in the absence of E1B.
    Quinlan MP.
    Oncogene; 1994 Sep 15; 9(9):2639-47. PubMed ID: 8058328
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  • 17. Essential role of Raf in Ras transformation and deregulation of matrix metalloproteinase expression in ovarian epithelial cells.
    Ulkü AS, Schäfer R, Der CJ.
    Mol Cancer Res; 2003 Dec 15; 1(14):1077-88. PubMed ID: 14707291
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  • 18. Transformation-resistant mos revertant is unable to activate MAP kinase kinase in response to v-mos or v-raf.
    Topol LZ, Marx M, Calothy G, Blair DG.
    Cell Growth Differ; 1995 Jan 15; 6(1):27-38. PubMed ID: 7718484
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  • 19. Raf and RhoA cooperate to transform intestinal epithelial cells and induce growth resistance to transforming growth factor beta.
    Du J, Jiang B, Coffey RJ, Barnard J.
    Mol Cancer Res; 2004 Apr 15; 2(4):233-41. PubMed ID: 15140945
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  • 20. Cellular functions of TC10, a Rho family GTPase: regulation of morphology, signal transduction and cell growth.
    Murphy GA, Solski PA, Jillian SA, Pérez de la Ossa P, D'Eustachio P, Der CJ, Rush MG.
    Oncogene; 1999 Jul 01; 18(26):3831-45. PubMed ID: 10445846
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