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Journal Abstract Search

370 related items for PubMed ID: 9558089

  • 1. Thymic alterations induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin are strictly dependent on aryl hydrocarbon receptor activation in hemopoietic cells.
    Staples JE, Murante FG, Fiore NC, Gasiewicz TA, Silverstone AE.
    J Immunol; 1998 Apr 15; 160(8):3844-54. PubMed ID: 9558089
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  • 4. Targeted deletion of the aryl hydrocarbon receptor in dendritic cells prevents thymic atrophy in response to dioxin.
    Beamer CA, Kreitinger JM, Cole SL, Shepherd DM.
    Arch Toxicol; 2019 Feb 15; 93(2):355-368. PubMed ID: 30499018
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  • 5. 2,3,7,8-tetrachlorodibenzo-p-dioxin causes alterations in lymphocyte development and thymic atrophy in hemopoietic chimeras generated from mice deficient in ARNT2.
    Laiosa MD, Lai ZW, Thurmond TS, Fiore NC, DeRossi C, Holdener BC, Gasiewicz TA, Silverstone AE.
    Toxicol Sci; 2002 Sep 15; 69(1):117-24. PubMed ID: 12215665
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  • 6. Dioxin-induced adseverin expression in the mouse thymus is strictly regulated and dependent on the aryl hydrocarbon receptor.
    Svensson C, Silverstone AE, Lai ZW, Lundberg K.
    Biochem Biophys Res Commun; 2002 Mar 15; 291(5):1194-200. PubMed ID: 11883943
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  • 9. Cytokine gene expression during ontogeny in murine thymus on activation of the aryl hydrocarbon receptor by 2,3,7,8-tetrachlorodibenzo-p-dioxin.
    Lai ZW, Hundeiker C, Gleichmann E, Esser C.
    Mol Pharmacol; 1997 Jul 15; 52(1):30-7. PubMed ID: 9224809
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  • 10. Thymocyte development in Ah-receptor-deficient mice is refractory to TCDD-inducible changes.
    Hundeiker C, Pineau T, Cassar G, Betensky RA, Gleichmann E, Esser C.
    Int J Immunopharmacol; 1999 Dec 15; 21(12):841-59. PubMed ID: 10606004
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  • 11. 2,3,7,8-Tetrachlorodibenzo-p-dioxin alters the differentiation of alloreactive CD8+ T cells toward a regulatory T cell phenotype by a mechanism that is dependent on aryl hydrocarbon receptor in CD4+ T cells.
    Funatake CJ, Marshall NB, Kerkvliet NI.
    J Immunotoxicol; 2008 Jan 15; 5(1):81-91. PubMed ID: 18382861
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  • 14. 2,3,7,8-Tetrachlorodibenzo-p-dioxin and diethylstilbestrol affect thymocytes at different stages of development in fetal thymus organ culture.
    Lai ZW, Fiore NC, Gasiewicz TA, Silverstone AE.
    Toxicol Appl Pharmacol; 1998 Apr 15; 149(2):167-77. PubMed ID: 9571985
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  • 15. Detection of a novel population of fetal thymocytes characterized by preferential emigration and a TCRgammadelta+ T cell fate after dioxin exposure.
    Majora M, Frericks M, Temchura V, Reichmann G, Esser C.
    Int Immunopharmacol; 2005 Nov 15; 5(12):1659-74. PubMed ID: 16102516
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  • 16. Activation of the aryl hydrocarbon receptor reduces the number of precursor and effector T cells, but preserves thymic CD4+CD25+Foxp3+ regulatory T cells.
    Schulz VJ, Smit JJ, Bol-Schoenmakers M, van Duursen MB, van den Berg M, Pieters RH.
    Toxicol Lett; 2012 Nov 30; 215(2):100-9. PubMed ID: 23041608
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  • 17. Skewed differentiation of thymocytes toward CD8 T cells by 2,3,7,8-tetrachlorodibenzo- p-dioxin requires activation of the extracellular signal-related kinase pathway.
    Tsukumo S, Iwata M, Tohyama C, Nohara K.
    Arch Toxicol; 2002 Jun 30; 76(5-6):335-43. PubMed ID: 12107651
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