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Pubmed for Handhelds

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Journal Abstract Search

180 related items for PubMed ID: 9565402

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  • 28. A sodium channel mutation causing epilepsy in man exhibits subtle defects in fast inactivation and activation in vitro.
    Alekov A, Rahman MM, Mitrovic N, Lehmann-Horn F, Lerche H.
    J Physiol; 2000 Dec 15; 529 Pt 3(Pt 3):533-9. PubMed ID: 11118488
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  • 29. Two mutations in the IV/S4-S5 segment of the human skeletal muscle Na+ channel disrupt fast and enhance slow inactivation.
    Alekov AK, Peter W, Mitrovic N, Lehmann-Horn F, Lerche H.
    Neurosci Lett; 2001 Jun 29; 306(3):173-6. PubMed ID: 11406323
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  • 31. Probing the outer vestibule of a sodium channel voltage sensor.
    Yang N, George AL, Horn R.
    Biophys J; 1997 Nov 29; 73(5):2260-8. PubMed ID: 9370423
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  • 35. Paroxysmal extreme pain disorder mutations within the D3/S4-S5 linker of Nav1.7 cause moderate destabilization of fast inactivation.
    Jarecki BW, Sheets PL, Jackson JO, Cummins TR.
    J Physiol; 2008 Sep 01; 586(17):4137-53. PubMed ID: 18599537
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  • 36. Implication of the C-terminal region of the alpha-subunit of voltage-gated sodium channels in fast inactivation.
    Deschênes I, Trottier E, Chahine M.
    J Membr Biol; 2001 Sep 15; 183(2):103-14. PubMed ID: 11562792
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  • 39. Channel cytoplasmic loops alter voltage-dependent sodium channel activation in an isoform-specific manner.
    Bennett ES.
    J Physiol; 2001 Sep 01; 535(Pt 2):371-81. PubMed ID: 11533130
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  • 40. Neutralization of gating charges in domain II of the sodium channel alpha subunit enhances voltage-sensor trapping by a beta-scorpion toxin.
    Cestèle S, Scheuer T, Mantegazza M, Rochat H, Catterall WA.
    J Gen Physiol; 2001 Sep 01; 118(3):291-302. PubMed ID: 11524459
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