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Journal Abstract Search

357 related items for PubMed ID: 9575197

  • 21. Functional dissection of the pro-apoptotic protein Bik. Heterodimerization with anti-apoptosis proteins is insufficient for induction of cell death.
    Elangovan B, Chinnadurai G.
    J Biol Chem; 1997 Sep 26; 272(39):24494-8. PubMed ID: 9305912
    [Abstract] [Full Text] [Related]

  • 22. Nuclear export of adenovirus E4orf6 protein is necessary for its ability to antagonize apoptotic activity of BH3-only proteins.
    Aoyagi M, Higashino F, Yasuda M, Takahashi A, Sawada Y, Totsuka Y, Kohgo T, Sano H, Kobayashi M, Shindoh M.
    Oncogene; 2003 Oct 09; 22(44):6919-27. PubMed ID: 14534539
    [Abstract] [Full Text] [Related]

  • 23. Sequence-specific dimerization of the transmembrane domain of the "BH3-only" protein BNIP3 in membranes and detergent.
    Sulistijo ES, Jaszewski TM, MacKenzie KR.
    J Biol Chem; 2003 Dec 19; 278(51):51950-6. PubMed ID: 14532263
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  • 24. TNF-alpha signals apoptosis through a bid-dependent conformational change in Bax that is inhibited by E1B 19K.
    Perez D, White E.
    Mol Cell; 2000 Jul 19; 6(1):53-63. PubMed ID: 10949027
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  • 28. BH3 domains of BH3-only proteins differentially regulate Bax-mediated mitochondrial membrane permeabilization both directly and indirectly.
    Kuwana T, Bouchier-Hayes L, Chipuk JE, Bonzon C, Sullivan BA, Green DR, Newmeyer DD.
    Mol Cell; 2005 Feb 18; 17(4):525-35. PubMed ID: 15721256
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  • 30. Bak and Bax function to limit adenovirus replication through apoptosis induction.
    Cuconati A, Degenhardt K, Sundararajan R, Anschel A, White E.
    J Virol; 2002 May 18; 76(9):4547-58. PubMed ID: 11932420
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  • 31. A novel adenovirus E1B19K-binding protein B5 inhibits apoptosis induced by Nip3 by forming a heterodimer through the C-terminal hydrophobic region.
    Ohi N, Tokunaga A, Tsunoda H, Nakano K, Haraguchi K, Oda K, Motoyama N, Nakajima T.
    Cell Death Differ; 1999 Apr 18; 6(4):314-25. PubMed ID: 10381623
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  • 32. Bcl-2 family member Bfl-1/A1 sequesters truncated bid to inhibit is collaboration with pro-apoptotic Bak or Bax.
    Werner AB, de Vries E, Tait SW, Bontjer I, Borst J.
    J Biol Chem; 2002 Jun 21; 277(25):22781-8. PubMed ID: 11929871
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  • 34. Nix and Nip3 form a subfamily of pro-apoptotic mitochondrial proteins.
    Chen G, Cizeau J, Vande Velde C, Park JH, Bozek G, Bolton J, Shi L, Dubik D, Greenberg A.
    J Biol Chem; 1999 Jan 01; 274(1):7-10. PubMed ID: 9867803
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  • 35. E1B 19K inhibits Fas-mediated apoptosis through FADD-dependent sequestration of FLICE.
    Perez D, White E.
    J Cell Biol; 1998 Jun 01; 141(5):1255-66. PubMed ID: 9606216
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  • 36. [BNIP3 as an atypical representative of the Bcl-2 protein family. Part 1: BNIP3, a regulator of non-apoptotic programmed cell death].
    Swoboda E, Strzadała L.
    Postepy Hig Med Dosw (Online); 2009 Sep 10; 63():409-17. PubMed ID: 19745227
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  • 37. Upregulation of BNIP3 and translocation to mitochondria mediates cyanide-induced apoptosis in cortical cells.
    Prabhakaran K, Li L, Zhang L, Borowitz JL, Isom GE.
    Neuroscience; 2007 Nov 30; 150(1):159-67. PubMed ID: 17980495
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  • 38. BNIP3 subfamily BH3-only proteins: mitochondrial stress sensors in normal and pathological functions.
    Chinnadurai G, Vijayalingam S, Gibson SB.
    Oncogene; 2008 Dec 30; 27 Suppl 1(Suppl 1):S114-27. PubMed ID: 19641497
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  • 39. Regulation of BNIP3 in normal and cancer cells.
    Lee H, Paik SG.
    Mol Cells; 2006 Feb 28; 21(1):1-6. PubMed ID: 16511341
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