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167 related items for PubMed ID: 9642682

  • 1. Advanced glycation endproducts in neurofilament conglomeration of motoneurons in familial and sporadic amyotrophic lateral sclerosis.
    Chou SM, Wang HS, Taniguchi A, Bucala R.
    Mol Med; 1998 May; 4(5):324-32. PubMed ID: 9642682
    [Abstract] [Full Text] [Related]

  • 2. Advanced glycation endproducts are deposited in neuronal hyaline inclusions: a study on familial amyotrophic lateral sclerosis with superoxide dismutase-1 mutation.
    Shibata N, Hirano A, Kato S, Nagai R, Horiuchi S, Komori T, Umahara T, Asayama K, Kobayashi M.
    Acta Neuropathol; 1999 Mar; 97(3):240-6. PubMed ID: 10090670
    [Abstract] [Full Text] [Related]

  • 3. A receptor for advanced glycosylation endproducts (AGEs) is colocalized with neurofilament-bound AGEs and SOD1 in motoneurons of ALS: immunohistochemical study.
    Chou SM, Han CY, Wang HS, Vlassara H, Bucala R.
    J Neurol Sci; 1999 Oct 31; 169(1-2):87-92. PubMed ID: 10540014
    [Abstract] [Full Text] [Related]

  • 4. Advanced glycation endproduct-modified superoxide dismutase-1 (SOD1)-positive inclusions are common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutations and transgenic mice expressing human SOD1 with a G85R mutation.
    Kato S, Horiuchi S, Liu J, Cleveland DW, Shibata N, Nakashima K, Nagai R, Hirano A, Takikawa M, Kato M, Nakano I, Ohama E.
    Acta Neuropathol; 2000 Nov 31; 100(5):490-505. PubMed ID: 11045671
    [Abstract] [Full Text] [Related]

  • 5. Amyotrophic lateral sclerosis associated with genetic abnormalities in the gene encoding Cu/Zn superoxide dismutase: molecular pathology of five new cases, and comparison with previous reports and 73 sporadic cases of ALS.
    Ince PG, Tomkins J, Slade JY, Thatcher NM, Shaw PJ.
    J Neuropathol Exp Neurol; 1998 Oct 31; 57(10):895-904. PubMed ID: 9786240
    [Abstract] [Full Text] [Related]

  • 6. Nonoxidative protein glycation is implicated in familial amyotrophic lateral sclerosis with superoxide dismutase-1 mutation.
    Shibata N, Nagai R, Miyata S, Jono T, Horiuchi S, Hirano A, Kato S, Sasaki S, Asayama K, Kobayashi M.
    Acta Neuropathol; 2000 Sep 31; 100(3):275-84. PubMed ID: 10965797
    [Abstract] [Full Text] [Related]

  • 7. Serpin=serine protease-like complexes within neurofilament conglomerates of motoneurons in amyotrophic lateral sclerosis.
    Chou SM, Taniguchi A, Wang HS, Festoff BW.
    J Neurol Sci; 1998 Oct 31; 160 Suppl 1():S73-9. PubMed ID: 9851654
    [Abstract] [Full Text] [Related]

  • 8. Colocalization of NOS and SOD1 in neurofilament accumulation within motor neurons of amyotrophic lateral sclerosis: an immunohistochemical study.
    Chou SM, Wang HS, Komai K.
    J Chem Neuroanat; 1996 Jun 31; 10(3-4):249-58. PubMed ID: 8811414
    [Abstract] [Full Text] [Related]

  • 9. Formation of advanced glycation end-product-modified superoxide dismutase-1 (SOD1) is one of the mechanisms responsible for inclusions common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutation, and transgenic mice expressing human SOD1 gene mutation.
    Kato S, Nakashima K, Horiuchi S, Nagai R, Cleveland DW, Liu J, Hirano A, Takikawa M, Kato M, Nakano I, Sakoda S, Asayama K, Ohama E.
    Neuropathology; 2001 Mar 31; 21(1):67-81. PubMed ID: 11304045
    [Abstract] [Full Text] [Related]

  • 10. HtrA2/Omi-immunoreactive intraneuronal inclusions in the anterior horn of patients with sporadic and Cu/Zn superoxide dismutase (SOD1) mutant amyotrophic lateral sclerosis.
    Kawamoto Y, Ito H, Kobayashi Y, Suzuki Y, Akiguchi I, Fujimura H, Sakoda S, Kusaka H, Hirano A, Takahashi R.
    Neuropathol Appl Neurobiol; 2010 Jun 31; 36(4):331-44. PubMed ID: 20202124
    [Abstract] [Full Text] [Related]

  • 11. Histological evidence of protein aggregation in mutant SOD1 transgenic mice and in amyotrophic lateral sclerosis neural tissues.
    Watanabe M, Dykes-Hoberg M, Culotta VC, Price DL, Wong PC, Rothstein JD.
    Neurobiol Dis; 2001 Dec 31; 8(6):933-41. PubMed ID: 11741389
    [Abstract] [Full Text] [Related]

  • 12. Increased 3-nitrotyrosine in both sporadic and familial amyotrophic lateral sclerosis.
    Beal MF, Ferrante RJ, Browne SE, Matthews RT, Kowall NW, Brown RH.
    Ann Neurol; 1997 Oct 31; 42(4):644-54. PubMed ID: 9382477
    [Abstract] [Full Text] [Related]

  • 13. Selective formation of certain advanced glycation end products in spinal cord astrocytes of humans and mice with superoxide dismutase-1 mutation.
    Shibata N, Hirano A, Hedley-Whyte ET, Dal Canto MC, Nagai R, Uchida K, Horiuchi S, Kawaguchi M, Yamamoto T, Kobayashi M.
    Acta Neuropathol; 2002 Aug 31; 104(2):171-8. PubMed ID: 12111360
    [Abstract] [Full Text] [Related]

  • 14. Astrocytic hyaline inclusions contain advanced glycation endproducts in familial amyotrophic lateral sclerosis with superoxide dismutase 1 gene mutation: immunohistochemical and immunoelectron microscopical analyses.
    Kato S, Horiuchi S, Nakashima K, Hirano A, Shibata N, Nakano I, Saito M, Kato M, Asayama K, Ohama E.
    Acta Neuropathol; 1999 Mar 31; 97(3):260-6. PubMed ID: 10090673
    [Abstract] [Full Text] [Related]

  • 15. Role of SOD-1 and nitric oxide/cyclic GMP cascade on neurofilament aggregation in ALS/MND.
    Chou SM, Wang HS, Taniguchi A.
    J Neurol Sci; 1996 Aug 31; 139 Suppl():16-26. PubMed ID: 8899653
    [Abstract] [Full Text] [Related]

  • 16. Amyotrophic lateral sclerosis is a non-amyloid disease in which extensive misfolding of SOD1 is unique to the familial form.
    Kerman A, Liu HN, Croul S, Bilbao J, Rogaeva E, Zinman L, Robertson J, Chakrabartty A.
    Acta Neuropathol; 2010 Mar 31; 119(3):335-44. PubMed ID: 20111867
    [Abstract] [Full Text] [Related]

  • 17. Accumulation of phosphorylated neurofilaments in anterior horn motoneurons of amyotrophic lateral sclerosis patients.
    Munoz DG, Greene C, Perl DP, Selkoe DJ.
    J Neuropathol Exp Neurol; 1988 Jan 31; 47(1):9-18. PubMed ID: 3334727
    [Abstract] [Full Text] [Related]

  • 18. The fragmented neuronal Golgi apparatus in amyotrophic lateral sclerosis includes the trans-Golgi-network: functional implications.
    Stieber A, Chen Y, Wei S, Mourelatos Z, Gonatas J, Okamoto K, Gonatas NK.
    Acta Neuropathol; 1998 Mar 31; 95(3):245-53. PubMed ID: 9542589
    [Abstract] [Full Text] [Related]

  • 19. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC.
    Neurobiol Dis; 2000 Dec 31; 7(6 Pt B):623-43. PubMed ID: 11114261
    [Abstract] [Full Text] [Related]

  • 20. Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutations.
    Mackenzie IR, Bigio EH, Ince PG, Geser F, Neumann M, Cairns NJ, Kwong LK, Forman MS, Ravits J, Stewart H, Eisen A, McClusky L, Kretzschmar HA, Monoranu CM, Highley JR, Kirby J, Siddique T, Shaw PJ, Lee VM, Trojanowski JQ.
    Ann Neurol; 2007 May 31; 61(5):427-34. PubMed ID: 17469116
    [Abstract] [Full Text] [Related]

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