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Journal Abstract Search

377 related items for PubMed ID: 9648840

  • 1. Functional analyses of novel mutations in the sulfonylurea receptor 1 associated with persistent hyperinsulinemic hypoglycemia of infancy.
    Shyng SL, Ferrigni T, Shepard JB, Nestorowicz A, Glaser B, Permutt MA, Nichols CG.
    Diabetes; 1998 Jul; 47(7):1145-51. PubMed ID: 9648840
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  • 2. Defective trafficking and function of KATP channels caused by a sulfonylurea receptor 1 mutation associated with persistent hyperinsulinemic hypoglycemia of infancy.
    Cartier EA, Conti LR, Vandenberg CA, Shyng SL.
    Proc Natl Acad Sci U S A; 2001 Feb 27; 98(5):2882-7. PubMed ID: 11226335
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  • 3. Genetic analysis of Japanese patients with persistent hyperinsulinemic hypoglycemia of infancy: nucleotide-binding fold-2 mutation impairs cooperative binding of adenine nucleotides to sulfonylurea receptor 1.
    Tanizawa Y, Matsuda K, Matsuo M, Ohta Y, Ochi N, Adachi M, Koga M, Mizuno S, Kajita M, Tanaka Y, Tachibana K, Inoue H, Furukawa S, Amachi T, Ueda K, Oka Y.
    Diabetes; 2000 Jan 27; 49(1):114-20. PubMed ID: 10615958
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  • 7. A point mutation inactivating the sulfonylurea receptor causes the severe form of persistent hyperinsulinemic hypoglycemia of infancy in Finland.
    Otonkoski T, Ammälä C, Huopio H, Cote GJ, Chapman J, Cosgrove K, Ashfield R, Huang E, Komulainen J, Ashcroft FM, Dunne MJ, Kere J, Thomas PM.
    Diabetes; 1999 Feb 27; 48(2):408-15. PubMed ID: 10334322
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  • 13. Loss of imprinted genes and paternal SUR1 mutations lead to hyperinsulinism in focal adenomatous hyperplasia.
    Fournet JC, Verkarre V, De Lonlay P, Rahier J, Brunelle F, Robert JJ, Nihoul-Fékété C, Saudubray JM, Junien C.
    Ann Endocrinol (Paris); 1998 Feb 27; 59(6):485-91. PubMed ID: 10189991
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  • 20. Molecular biology of adenosine triphosphate-sensitive potassium channels.
    Aguilar-Bryan L, Bryan J.
    Endocr Rev; 1999 Apr 27; 20(2):101-35. PubMed ID: 10204114
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