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28. The structure and function of the ATP-sensitive K+ channel in insulin-secreting pancreatic beta-cells. Miki T, Nagashima K, Seino S. J Mol Endocrinol; 1999 Apr 12; 22(2):113-23. PubMed ID: 10194514 [Abstract] [Full Text] [Related]
30. Regulation of cloned ATP-sensitive K channels by phosphorylation, MgADP, and phosphatidylinositol bisphosphate (PIP(2)): a study of channel rundown and reactivation. Ribalet B, John SA, Weiss JN. J Gen Physiol; 2000 Sep 12; 116(3):391-410. PubMed ID: 10962016 [Abstract] [Full Text] [Related]
35. Severe congenital hyperinsulinism caused by a mutation in the Kir6.2 subunit of the adenosine triphosphate-sensitive potassium channel impairing trafficking and function. Marthinet E, Bloc A, Oka Y, Tanizawa Y, Wehrle-Haller B, Bancila V, Dubuis JM, Philippe J, Schwitzgebel VM. J Clin Endocrinol Metab; 2005 Sep 27; 90(9):5401-6. PubMed ID: 15998776 [Abstract] [Full Text] [Related]
36. Carriers of an inactivating beta-cell ATP-sensitive K(+) channel mutation have normal glucose tolerance and insulin sensitivity and appropriate insulin secretion. Huopio H, Vauhkonen I, Komulainen J, Niskanen L, Otonkoski T, Laakso M. Diabetes Care; 2002 Jan 27; 25(1):101-6. PubMed ID: 11772909 [Abstract] [Full Text] [Related]
38. Genotype-phenotype correlations in children with congenital hyperinsulinism due to recessive mutations of the adenosine triphosphate-sensitive potassium channel genes. Henwood MJ, Kelly A, Macmullen C, Bhatia P, Ganguly A, Thornton PS, Stanley CA. J Clin Endocrinol Metab; 2005 Feb 27; 90(2):789-94. PubMed ID: 15562009 [Abstract] [Full Text] [Related]