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445 related items for PubMed ID: 9661886

  • 1. Ornithine decarboxylase induction in transformation by H-Ras and RhoA.
    Shantz LM, Pegg AE.
    Cancer Res; 1998 Jul 01; 58(13):2748-53. PubMed ID: 9661886
    [Abstract] [Full Text] [Related]

  • 2. Raf and RhoA cooperate to transform intestinal epithelial cells and induce growth resistance to transforming growth factor beta.
    Du J, Jiang B, Coffey RJ, Barnard J.
    Mol Cancer Res; 2004 Apr 01; 2(4):233-41. PubMed ID: 15140945
    [Abstract] [Full Text] [Related]

  • 3. Rit, a non-lipid-modified Ras-related protein, transforms NIH3T3 cells without activating the ERK, JNK, p38 MAPK or PI3K/Akt pathways.
    Rusyn EV, Reynolds ER, Shao H, Grana TM, Chan TO, Andres DA, Cox AD.
    Oncogene; 2000 Sep 28; 19(41):4685-94. PubMed ID: 11032018
    [Abstract] [Full Text] [Related]

  • 4. Expression of an ornithine decarboxylase dominant-negative mutant reverses eukaryotic initiation factor 4E-induced cell transformation.
    Shantz LM, Coleman CS, Pegg AE.
    Cancer Res; 1996 Nov 15; 56(22):5136-40. PubMed ID: 8912847
    [Abstract] [Full Text] [Related]

  • 5. Elevated levels of ornithine decarboxylase cooperate with Raf/ERK activation to convert normal keratinocytes into invasive malignant cells.
    Hayes CS, DeFeo K, Lan L, Paul B, Sell C, Gilmour SK.
    Oncogene; 2006 Mar 09; 25(10):1543-53. PubMed ID: 16278677
    [Abstract] [Full Text] [Related]

  • 6. Essential role of Raf in Ras transformation and deregulation of matrix metalloproteinase expression in ovarian epithelial cells.
    Ulkü AS, Schäfer R, Der CJ.
    Mol Cancer Res; 2003 Dec 09; 1(14):1077-88. PubMed ID: 14707291
    [Abstract] [Full Text] [Related]

  • 7. Retroviral gene transfer of dominant negative raf-1 mutants suppresses ha-ras-induced transformation and delays tumor formation.
    Heinicke T, Radziwill G, Nawrath M, Rommel C, Pavlovic J, Moelling K.
    Cancer Gene Ther; 2000 May 09; 7(5):697-706. PubMed ID: 10830717
    [Abstract] [Full Text] [Related]

  • 8. Tumour promoter mediated altered expression and regulation of ornithine decarboxylase and S-adenosylmethionine decarboxylase in H-ras-transformed fibrosarcoma cell lines.
    Voskas D, Mader R, Lee J, Hurta RA.
    Biochem Cell Biol; 2001 May 09; 79(1):69-81. PubMed ID: 11235918
    [Abstract] [Full Text] [Related]

  • 9. Transformation by H-ras can result in aberrant regulation of ornithine decarboxylase gene expression by transforming growth factor-beta(1).
    Hurta RA, Lee J, Voskas D.
    J Cell Biochem; 2001 May 09; 81(1):39-55. PubMed ID: 11180396
    [Abstract] [Full Text] [Related]

  • 10. Signal transduction elements of TC21, an oncogenic member of the R-Ras subfamily of GTP-binding proteins.
    Movilla N, Crespo P, Bustelo XR.
    Oncogene; 1999 Oct 21; 18(43):5860-9. PubMed ID: 10557073
    [Abstract] [Full Text] [Related]

  • 11. Protein kinase Cepsilon is oncogenic in colon epithelial cells by interaction with the ras signal transduction pathway.
    Perletti GP, Concari P, Brusaferri S, Marras E, Piccinini F, Tashjian AH.
    Oncogene; 1998 Jun 25; 16(25):3345-8. PubMed ID: 9681835
    [Abstract] [Full Text] [Related]

  • 12. Ras mediates radioresistance through both phosphatidylinositol 3-kinase-dependent and Raf-dependent but mitogen-activated protein kinase/extracellular signal-regulated kinase kinase-independent signaling pathways.
    Grana TM, Rusyn EV, Zhou H, Sartor CI, Cox AD.
    Cancer Res; 2002 Jul 15; 62(14):4142-50. PubMed ID: 12124353
    [Abstract] [Full Text] [Related]

  • 13. Differential regulation of cyclooxygenase 2 expression by small GTPases Ras, Rac1, and RhoA.
    Chang YW, Putzer K, Ren L, Kaboord B, Chance TW, Qoronfleh MW, Jakobi R.
    J Cell Biochem; 2005 Oct 01; 96(2):314-29. PubMed ID: 16088958
    [Abstract] [Full Text] [Related]

  • 14. Ornithine decarboxylase gene expression is aberrantly regulated via the cAMP signal transduction pathway in malignant H-ras transformed cell lines.
    Hurta RA, Wright JA.
    J Cell Physiol; 1994 Nov 01; 161(2):383-91. PubMed ID: 7525612
    [Abstract] [Full Text] [Related]

  • 15. Transformation of NIH/3T3 cells by ornithine decarboxylase overexpression.
    Moshier JA, Dosescu J, Skunca M, Luk GD.
    Cancer Res; 1993 Jun 01; 53(11):2618-22. PubMed ID: 8495425
    [Abstract] [Full Text] [Related]

  • 16. TC21 and Ras share indistinguishable transforming and differentiating activities.
    Graham SM, Oldham SM, Martin CB, Drugan JK, Zohn IE, Campbell S, Der CJ.
    Oncogene; 1999 Mar 25; 18(12):2107-16. PubMed ID: 10321735
    [Abstract] [Full Text] [Related]

  • 17. Ras-interacting domain of Ral GDP dissociation stimulator like (RGL) reverses v-Ras-induced transformation and Raf-1 activation in NIH3T3 cells.
    Okazaki M, Kishida S, Murai H, Hinoi T, Kikuchi A.
    Cancer Res; 1996 May 15; 56(10):2387-92. PubMed ID: 8625316
    [Abstract] [Full Text] [Related]

  • 18. R-Ras is regulated by activators and effectors distinct from those that control Ras function.
    Huff SY, Quilliam LA, Cox AD, Der CJ.
    Oncogene; 1997 Jan 16; 14(2):133-43. PubMed ID: 9010215
    [Abstract] [Full Text] [Related]

  • 19. Endothelial exposure to hypoxia induces Egr-1 expression involving PKCalpha-mediated Ras/Raf-1/ERK1/2 pathway.
    Lo LW, Cheng JJ, Chiu JJ, Wung BS, Liu YC, Wang DL.
    J Cell Physiol; 2001 Sep 16; 188(3):304-12. PubMed ID: 11473356
    [Abstract] [Full Text] [Related]

  • 20. A role for Ras in v-Crk transformation.
    Greulich H, Hanafusa H.
    Cell Growth Differ; 1996 Nov 16; 7(11):1443-51. PubMed ID: 8930393
    [Abstract] [Full Text] [Related]

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