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Journal Abstract Search

157 related items for PubMed ID: 9685222

  • 1. Primary culture of cells from hyperfunctioning thyroid adenoma with an activating mutation of G alphas.
    Kamiya Y, Murakami M, Yanagita Y, Koitabashi H, Nagamachi Y, Hosoi Y, Ogiwara T, Mizuma H, Iriuchijima T, Mori M.
    Mol Cell Endocrinol; 1998 Mar 16; 138(1-2):137-42. PubMed ID: 9685222
    [Abstract] [Full Text] [Related]

  • 2. Thyrotropin receptor gene alterations in thyroid hyperfunctioning adenomas.
    Russo D, Arturi F, Suarez HG, Schlumberger M, Du Villard JA, Crocetti U, Filetti S.
    J Clin Endocrinol Metab; 1996 Apr 16; 81(4):1548-51. PubMed ID: 8636365
    [Abstract] [Full Text] [Related]

  • 3. Diversity and prevalence of somatic mutations in the thyrotropin receptor and Gs alpha genes as a cause of toxic thyroid adenomas.
    Parma J, Duprez L, Van Sande J, Hermans J, Rocmans P, Van Vliet G, Costagliola S, Rodien P, Dumont JE, Vassart G.
    J Clin Endocrinol Metab; 1997 Aug 16; 82(8):2695-701. PubMed ID: 9253356
    [Abstract] [Full Text] [Related]

  • 4. Functioning and nonfunctioning thyroid adenomas involve different molecular pathogenetic mechanisms.
    Tonacchera M, Vitti P, Agretti P, Ceccarini G, Perri A, Cavaliere R, Mazzi B, Naccarato AG, Viacava P, Miccoli P, Pinchera A, Chiovato L.
    J Clin Endocrinol Metab; 1999 Nov 16; 84(11):4155-8. PubMed ID: 10566665
    [Abstract] [Full Text] [Related]

  • 5. Mutations of thyrotropin receptor isolated from thyroid autonomous functioning adenomas confer TSH-independent growth to thyroid cells.
    Porcellini A, Ruggiano G, Pannain S, Ciullo I, Amabile G, Fenzi G, Avvedimento EV.
    Oncogene; 1997 Aug 14; 15(7):781-9. PubMed ID: 9266964
    [Abstract] [Full Text] [Related]

  • 6. The 3',5'-cyclic adenosine monophosphate response element binding protein (CREB) is functionally reduced in human toxic thyroid adenomas.
    Brunetti A, Chiefari E, Filetti S, Russo D.
    Endocrinology; 2000 Feb 14; 141(2):722-30. PubMed ID: 10650954
    [Abstract] [Full Text] [Related]

  • 7. Somatic mutations in the VI transmembrane segment of the thyrotropin receptor constitutively activate cAMP signalling in thyroid hyperfunctioning adenomas.
    Porcellini A, Ciullo I, Pannain S, Fenzi G, Avvedimento E.
    Oncogene; 1995 Sep 21; 11(6):1089-93. PubMed ID: 7566968
    [Abstract] [Full Text] [Related]

  • 8. Thyroid hyperfunctioning adenomas with and without Gsp/TSH receptor mutations show similar clinical features.
    Arturi F, Capula C, Chiefari E, Filetti S, Russo D.
    Exp Clin Endocrinol Diabetes; 1998 Sep 21; 106(3):234-6. PubMed ID: 9710366
    [Abstract] [Full Text] [Related]

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  • 10. Hyperfunctioning thyroid nodules in toxic multinodular goiter share activating thyrotropin receptor mutations with solitary toxic adenoma.
    Tonacchera M, Chiovato L, Pinchera A, Agretti P, Fiore E, Cetani F, Rocchi R, Viacava P, Miccoli P, Vitti P.
    J Clin Endocrinol Metab; 1998 Feb 21; 83(2):492-8. PubMed ID: 9467563
    [Abstract] [Full Text] [Related]

  • 11. Expression of cAMP-responsive element binding protein and inducible cAMP early repressor in hyperfunctioning thyroid adenomas.
    Peri A, Luciani P, Tonacchera M, Agretti P, Baglioni-Peri S, Buci L, Conforti B, Cioppi F, Biliotti G, Serio M, Vitti P, Chiovato L.
    Eur J Endocrinol; 2002 Jun 21; 146(6):759-66. PubMed ID: 12039695
    [Abstract] [Full Text] [Related]

  • 12. Somatic mutations in the thyrotropin receptor gene cause hyperfunctioning thyroid adenomas.
    Parma J, Duprez L, Van Sande J, Cochaux P, Gervy C, Mockel J, Dumont J, Vassart G.
    Nature; 1993 Oct 14; 365(6447):649-51. PubMed ID: 8413627
    [Abstract] [Full Text] [Related]

  • 13. Structural studies of the thyrotropin receptor and Gs alpha in human thyroid cancers: low prevalence of mutations predicts infrequent involvement in malignant transformation.
    Spambalg D, Sharifi N, Elisei R, Gross JL, Medeiros-Neto G, Fagin JA.
    J Clin Endocrinol Metab; 1996 Nov 14; 81(11):3898-901. PubMed ID: 8923835
    [Abstract] [Full Text] [Related]

  • 14. Expression of G(alpha)(s) proteins and TSH receptor signalling in hyperfunctioning thyroid nodules with TSH receptor mutations.
    Holzapfel HP, Bergner B, Wonerow P, Paschke R.
    Eur J Endocrinol; 2002 Jul 14; 147(1):109-16. PubMed ID: 12088927
    [Abstract] [Full Text] [Related]

  • 15. Gs alpha mutations in hyperfunctioning thyroid adenomas.
    Murakami M, Kamiya Y, Yanagita Y, Mori M.
    Arch Med Res; 1999 Jul 14; 30(6):514-21. PubMed ID: 10714366
    [Abstract] [Full Text] [Related]

  • 16. Identification and functional characterization of two new somatic mutations causing constitutive activation of the thyrotropin receptor in hyperfunctioning autonomous adenomas of the thyroid.
    Paschke R, Tonacchera M, Van Sande J, Parma J, Vassart G.
    J Clin Endocrinol Metab; 1994 Dec 14; 79(6):1785-9. PubMed ID: 7989485
    [Abstract] [Full Text] [Related]

  • 17. Thyrotropin receptor mutations in hyperfunctioning thyroid adenomas from Brazil.
    Nogueira CR, Kopp P, Arseven OK, Santos CL, Jameson JL, Medeiros-Neto G.
    Thyroid; 1999 Nov 14; 9(11):1063-8. PubMed ID: 10595453
    [Abstract] [Full Text] [Related]

  • 18. Activating mutations of the TSH receptor in differentiated thyroid carcinomas.
    Russo D, Arturi F, Schlumberger M, Caillou B, Monier R, Filetti S, Suárez HG.
    Oncogene; 1995 Nov 02; 11(9):1907-11. PubMed ID: 7478621
    [Abstract] [Full Text] [Related]

  • 19. Congenital hyperthyroidism caused by a solitary toxic adenoma harboring a novel somatic mutation (serine281-->isoleucine) in the extracellular domain of the thyrotropin receptor.
    Kopp P, Muirhead S, Jourdain N, Gu WX, Jameson JL, Rodd C.
    J Clin Invest; 1997 Sep 15; 100(6):1634-9. PubMed ID: 9294132
    [Abstract] [Full Text] [Related]

  • 20. Identification and functional characterization of two novel activating thyrotropin receptor mutants in toxic thyroid follicular adenomas.
    Castro I, Lima L, Seoane R, Lado-Abeal J.
    Thyroid; 2009 Jun 15; 19(6):645-9. PubMed ID: 19499991
    [Abstract] [Full Text] [Related]

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