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31. Elevated free nitrotyrosine levels, but not protein-bound nitrotyrosine or hydroxyl radicals, throughout amyotrophic lateral sclerosis (ALS)-like disease implicate tyrosine nitration as an aberrant in vivo property of one familial ALS-linked superoxide dismutase 1 mutant. Bruijn LI, Beal MF, Becher MW, Schulz JB, Wong PC, Price DL, Cleveland DW. Proc Natl Acad Sci U S A; 1997 Jul 08; 94(14):7606-11. PubMed ID: 9207139 [Abstract] [Full Text] [Related]
32. Amyotrophic lateral sclerosis associated with genetic abnormalities in the gene encoding Cu/Zn superoxide dismutase: molecular pathology of five new cases, and comparison with previous reports and 73 sporadic cases of ALS. Ince PG, Tomkins J, Slade JY, Thatcher NM, Shaw PJ. J Neuropathol Exp Neurol; 1998 Oct 08; 57(10):895-904. PubMed ID: 9786240 [Abstract] [Full Text] [Related]
38. Massive mitochondrial degeneration in motor neurons triggers the onset of amyotrophic lateral sclerosis in mice expressing a mutant SOD1. Kong J, Xu Z. J Neurosci; 1998 May 01; 18(9):3241-50. PubMed ID: 9547233 [Abstract] [Full Text] [Related]
40. Mutant SOD1 in cell types other than motor neurons and oligodendrocytes accelerates onset of disease in ALS mice. Yamanaka K, Boillee S, Roberts EA, Garcia ML, McAlonis-Downes M, Mikse OR, Cleveland DW, Goldstein LS. Proc Natl Acad Sci U S A; 2008 May 27; 105(21):7594-9. PubMed ID: 18492803 [Abstract] [Full Text] [Related] Page: [Previous] [Next] [New Search]