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27. Similar peptides from two beta cell autoantigens, proinsulin and glutamic acid decarboxylase, stimulate T cells of individuals at risk for insulin-dependent diabetes. Rudy G, Stone N, Harrison LC, Colman PG, McNair P, Brusic V, French MB, Honeyman MC, Tait B, Lew AM. Mol Med; 1995 Sep; 1(6):625-33. PubMed ID: 8529129 [Abstract] [Full Text] [Related]
28. Antibodies to glutamic acid decarboxylase and insulin-dependent diabetes in patients with autoimmune polyendocrine syndrome type I. Tuomi T, Björses P, Falorni A, Partanen J, Perheentupa J, Lernmark A, Miettinen A. J Clin Endocrinol Metab; 1996 Apr; 81(4):1488-94. PubMed ID: 8636356 [Abstract] [Full Text] [Related]
29. Human islet cell antibodies: immunoglobulin class and subclass distribution defined by monoclonal antibodies. Omar MA, Srikanta S, Eisenbarth GS. Diabetes Res; 1987 Apr; 4(4):155-7. PubMed ID: 3304781 [Abstract] [Full Text] [Related]
30. Human B cells secreting immunoglobulin G to glutamic acid decarboxylase-65 from a nondiabetic patient with multiple autoantibodies and Graves' disease: a comparison with those present in type 1 diabetes. Tremble J, Morgenthaler NG, Vlug A, Powers AC, Christie MR, Scherbaum WA, Banga JP. J Clin Endocrinol Metab; 1997 Aug; 82(8):2664-70. PubMed ID: 9253351 [Abstract] [Full Text] [Related]
31. Associations of anti-GAD antibodies with islet cell antibodies and insulin autoantibodies in first-degree relatives of type I diabetic patients. Roll U, Christie MR, Standl E, Ziegler AG. Diabetes; 1994 Jan; 43(1):154-60. PubMed ID: 8262311 [Abstract] [Full Text] [Related]
32. [Evaluation of the importance of antibodies against islet cells (ICA) and Glutamate Decarboxylase(anti-GAD) for determining prognosis of diabetes type 1 in the Polish population--5 year observation]. Kretowski A, Szelachowska M, Kinalska I. Pol Arch Med Wewn; 1999 Apr; 101(4):323-7. PubMed ID: 10740410 [Abstract] [Full Text] [Related]
33. Human monoclonal islet cell antibodies from a patient with insulin-dependent diabetes mellitus reveal glutamate decarboxylase as the target antigen. Richter W, Endl J, Eiermann TH, Brandt M, Kientsch-Engel R, Thivolet C, Jungfer H, Scherbaum WA. Proc Natl Acad Sci U S A; 1992 Sep 15; 89(18):8467-71. PubMed ID: 1382289 [Abstract] [Full Text] [Related]
34. Autoantibodies to glutamic acid decarboxylase (GAD) detected by an immuno-trapping enzyme activity assay: relation to insulin-dependent diabetes mellitus and islet cell antibodies. Martino GV, Tappaz ML, Braghi S, Dozio N, Canal N, Pozza G, Bottazzo GF, Grimaldi LM, Bosi E. J Autoimmun; 1991 Dec 15; 4(6):915-23. PubMed ID: 1812896 [Abstract] [Full Text] [Related]
35. Autoantibodies against glutamic acid decarboxylase 65 in Japanese patients with insulin-dependent diabetes mellitus (IDDM). Yano M, Moriuchi R, Kawasaki E, Watanabe M, Saito K, Takino H, Akazawa S, Miyamoto T, Nagataki S. J Autoimmun; 1995 Feb 15; 8(1):83-96. PubMed ID: 7734039 [Abstract] [Full Text] [Related]
37. The combination of antibodies to GAD-65 and IA-2ic can replace the islet-cell antibody assay to identify subjects at risk of type 1 diabetes mellitus. Hatziagelaki E, Jaeger C, Petzoldt R, Seissler J, Scherbaum WA, Federlin K, Bretzel RG. Horm Metab Res; 1999 Oct 15; 31(10):564-9. PubMed ID: 10596966 [Abstract] [Full Text] [Related]
38. Persistent GAD 65 antibodies in longstanding IDDM are not associated with residual beta-cell function, neuropathy or HLA-DR status. Jaeger C, Allendörfer J, Hatziagelaki E, Dyrberg T, Bergis KH, Federlin K, Bretzel RG. Horm Metab Res; 1997 Oct 15; 29(10):510-5. PubMed ID: 9405979 [Abstract] [Full Text] [Related]