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Journal Abstract Search

237 related items for PubMed ID: 9742125

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  • 24. The conserved N-terminal BH4 domain of Bcl-2 homologues is essential for inhibition of apoptosis and interaction with CED-4.
    Huang DC, Adams JM, Cory S.
    EMBO J; 1998 Feb 16; 17(4):1029-39. PubMed ID: 9463381
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  • 25. Interaction of E1B 19K with Bax is required to block Bax-induced loss of mitochondrial membrane potential and apoptosis.
    Han J, Modha D, White E.
    Oncogene; 1998 Dec 10; 17(23):2993-3005. PubMed ID: 9881701
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  • 26. BH3 domain of BAD is required for heterodimerization with BCL-XL and pro-apoptotic activity.
    Zha J, Harada H, Osipov K, Jockel J, Waksman G, Korsmeyer SJ.
    J Biol Chem; 1997 Sep 26; 272(39):24101-4. PubMed ID: 9305851
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  • 28. Diva, a Bcl-2 homologue that binds directly to Apaf-1 and induces BH3-independent cell death.
    Inohara N, Gourley TS, Carrio R, Muñiz M, Merino J, Garcia I, Koseki T, Hu Y, Chen S, Núñez G.
    J Biol Chem; 1998 Dec 04; 273(49):32479-86. PubMed ID: 9829980
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  • 29. BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis.
    Cheng EH, Wei MC, Weiler S, Flavell RA, Mak TW, Lindsten T, Korsmeyer SJ.
    Mol Cell; 2001 Sep 04; 8(3):705-11. PubMed ID: 11583631
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  • 33. Inhibition of Bax-induced cytochrome c release from neural cell and brain mitochondria by dibucaine and propranolol.
    Polster BM, Basañez G, Young M, Suzuki M, Fiskum G.
    J Neurosci; 2003 Apr 01; 23(7):2735-43. PubMed ID: 12684459
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  • 34. Structure-function analysis of Bcl-2 family proteins. Regulators of programmed cell death.
    Reed JC, Zha H, Aime-Sempe C, Takayama S, Wang HG.
    Adv Exp Med Biol; 1996 Apr 01; 406():99-112. PubMed ID: 8910675
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  • 35. Bax activation by the BH3-only protein Puma promotes cell dependence on antiapoptotic Bcl-2 family members.
    Gallenne T, Gautier F, Oliver L, Hervouet E, Noël B, Hickman JA, Geneste O, Cartron PF, Vallette FM, Manon S, Juin P.
    J Cell Biol; 2009 Apr 20; 185(2):279-90. PubMed ID: 19380879
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  • 36. Induction of cell death by the BH3-only Bcl-2 homolog Nbk/Bik is mediated by an entirely Bax-dependent mitochondrial pathway.
    Gillissen B, Essmann F, Graupner V, Stärck L, Radetzki S, Dörken B, Schulze-Osthoff K, Daniel PT.
    EMBO J; 2003 Jul 15; 22(14):3580-90. PubMed ID: 12853473
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  • 37. Functional dissection of the pro-apoptotic protein Bik. Heterodimerization with anti-apoptosis proteins is insufficient for induction of cell death.
    Elangovan B, Chinnadurai G.
    J Biol Chem; 1997 Sep 26; 272(39):24494-8. PubMed ID: 9305912
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  • 38. Bcl-XL protects BimEL-induced Bax conformational change and cytochrome C release independent of interacting with Bax or BimEL.
    Yamaguchi H, Wang HG.
    J Biol Chem; 2002 Nov 01; 277(44):41604-12. PubMed ID: 12198137
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  • 39. Targeting Bax interaction sites reveals that only homo-oligomerization sites are essential for its activation.
    Peng R, Tong JS, Li H, Yue B, Zou F, Yu J, Zhang L.
    Cell Death Differ; 2013 May 01; 20(5):744-54. PubMed ID: 23392123
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  • 40. Structural biology of the Bcl-2 family of proteins.
    Petros AM, Olejniczak ET, Fesik SW.
    Biochim Biophys Acta; 2004 Mar 01; 1644(2-3):83-94. PubMed ID: 14996493
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