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Journal Abstract Search

207 related items for PubMed ID: 9753873

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  • 3. Slow Ca2+ channels neither contribute to upstroke of action potential nor to pacemaker potential in spontaneously active freshly isolated three day embryonic chick ventricle.
    Prakash P, Tripathi O.
    Indian J Physiol Pharmacol; 2006; 50(2):121-32. PubMed ID: 17051731
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  • 4. The neuroprotective agent sipatrigine blocks multiple cardiac ion channels and causes triangulation of the ventricular action potential.
    Gao Z, Milnes JT, Choisy SC, Leach MJ, Hancox JC, James AF.
    Clin Exp Pharmacol Physiol; 2005 Dec; 32(12):1088-96. PubMed ID: 16445575
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  • 5. Effects of verapamil on rapid Na channel-dependent action potentials of K+-depolarized ventricular fibers.
    Chen CM, Gettes LS.
    J Pharmacol Exp Ther; 1979 Jun; 209(3):415-21. PubMed ID: 439018
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  • 6. Development of slow Ca2+-Na+ channels during organ culture of young embryonic chick hearts.
    Kojima M, Sperelakis N.
    J Dev Physiol; 1985 Dec; 7(6):355-63. PubMed ID: 2416799
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  • 7. Effects of 5-(N,N-hexamethylene)amiloride on action potentials, intracellular Na, and pH of guinea pig ventricular muscle in vitro.
    Lai ZF, Hotokebuchi N, Cragoe EJ, Nishi K.
    J Cardiovasc Pharmacol; 1994 Feb; 23(2):259-67. PubMed ID: 7511756
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  • 8. [T-channels and Na+,Ca2+-exchangers as components of the Ca2+-system of the myocardial activity regulation of the frog Rana temporaria].
    Shemarova IV, Kuznetsov SV, Demina IN, Nesterov VP.
    Zh Evol Biokhim Fiziol; 2009 Feb; 45(3):319-28. PubMed ID: 19569558
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  • 9. Fish cardiac sodium channels are tetrodotoxin sensitive.
    Haverinen J, Hassinen M, Vornanen M.
    Acta Physiol (Oxf); 2007 Nov; 191(3):197-204. PubMed ID: 17935523
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  • 10. Electrophysiologic effects of ketanserin on canine Purkinje fibers, ventricular myocardium and the intact heart.
    Zaza A, Malfatto G, Rosen MR.
    J Pharmacol Exp Ther; 1989 Jul; 250(1):397-405. PubMed ID: 2746507
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  • 11. Competitive inhibition of cardiac sodium channels by aprindine and lidocaine studied using a maximum upstroke velocity of action potential in guinea pig ventricular muscles.
    Kodama I, Toyama J, Yamada K.
    J Pharmacol Exp Ther; 1987 Jun; 241(3):1065-71. PubMed ID: 2439681
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  • 12. Apamin, a highly potent blocker of the TTX- and Mn2(+)-insensitive fast transient Na+ current in young embryonic heart.
    Bkaily G, Jacques D, Sculptoreanu A, Yamamoto T, Carrier D, Vigneault D, Sperelakis N.
    J Mol Cell Cardiol; 1991 Jan; 23(1):25-39. PubMed ID: 2038068
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  • 13. A comparative study of the blockade of calcium-dependent action potentials by verapamil, nifedipine and nimodipine in ventricular muscle.
    Hachisu M, Pappano AJ.
    J Pharmacol Exp Ther; 1983 Apr; 225(1):112-20. PubMed ID: 6834265
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  • 14. Block of avian cardiac fast sodium channels by tetrodotoxin is enhanced by repetitive depolarization but not by steady depolarization.
    Inoue D, Pappano AJ.
    J Mol Cell Cardiol; 1984 Oct; 16(10):943-52. PubMed ID: 6096567
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  • 15. Electrophysiological characterization of class III activity of a verapamil derivative in guinea-pig cardiac tissues.
    Kammer T, Berger F, Borchard U, Hafner D.
    Arzneimittelforschung; 1993 Mar; 43(3):302-8. PubMed ID: 8489557
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  • 16. Comparative effects of ultra-short-acting beta1-blockers on voltage-gated tetrodotoxin-resistant Na+ channels in rat sensory neurons.
    Tanahashi S, Iida H, Dohi S, Oda A, Osawa Y, Yamaguchi S.
    Eur J Anaesthesiol; 2009 Mar; 26(3):196-200. PubMed ID: 19237982
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  • 17. Role of TTX-sensitive and TTX-resistant sodium channels in Adelta- and C-fiber conduction and synaptic transmission.
    Pinto V, Derkach VA, Safronov BV.
    J Neurophysiol; 2008 Feb; 99(2):617-28. PubMed ID: 18057109
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  • 18. Effects of sodium deoxycholate on the mechanical and electrical activities and ultrastructure of guinea pig atria.
    Tripathi O, Ray M, Bajpai VK, Bhatnagar A, Kumar R, Singh YK, Singh C, Shipstone AC, Dhawan BN.
    Biomed Biochim Acta; 1988 Feb; 47(9):901-14. PubMed ID: 3248126
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  • 19. Modulation of flecainide's cardiac sodium channel blocking actions by extracellular sodium: a possible cellular mechanism for the action of sodium salts in flecainide cardiotoxicity.
    Ranger S, Sheldon R, Fermini B, Nattel S.
    J Pharmacol Exp Ther; 1993 Mar; 264(3):1160-7. PubMed ID: 8383739
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