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310 related items for PubMed ID: 9763433

  • 1. Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions.
    Eskes R, Antonsson B, Osen-Sand A, Montessuit S, Richter C, Sadoul R, Mazzei G, Nichols A, Martinou JC.
    J Cell Biol; 1998 Oct 05; 143(1):217-24. PubMed ID: 9763433
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  • 2. Bid-induced cytochrome c release is mediated by a pathway independent of mitochondrial permeability transition pore and Bax.
    Kim TH, Zhao Y, Barber MJ, Kuharsky DK, Yin XM.
    J Biol Chem; 2000 Dec 15; 275(50):39474-81. PubMed ID: 10982793
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  • 3. Functional consequences of the sustained or transient activation by Bax of the mitochondrial permeability transition pore.
    Pastorino JG, Tafani M, Rothman RJ, Marcinkeviciute A, Hoek JB, Farber JL.
    J Biol Chem; 1999 Oct 29; 274(44):31734-9. PubMed ID: 10531385
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  • 5. Proapoptotic BH3-only Bcl-2 family members induce cytochrome c release, but not mitochondrial membrane potential loss, and do not directly modulate voltage-dependent anion channel activity.
    Shimizu S, Tsujimoto Y.
    Proc Natl Acad Sci U S A; 2000 Jan 18; 97(2):577-82. PubMed ID: 10639121
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  • 7. Bax directly induces release of cytochrome c from isolated mitochondria.
    Jürgensmeier JM, Xie Z, Deveraux Q, Ellerby L, Bredesen D, Reed JC.
    Proc Natl Acad Sci U S A; 1998 Apr 28; 95(9):4997-5002. PubMed ID: 9560217
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  • 9. Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria.
    Narita M, Shimizu S, Ito T, Chittenden T, Lutz RJ, Matsuda H, Tsujimoto Y.
    Proc Natl Acad Sci U S A; 1998 Dec 08; 95(25):14681-6. PubMed ID: 9843949
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  • 12. Role of the permeability transition pore in cytochrome C release from mitochondria during ischemia-reperfusion in rat liver.
    Morin D, Pires F, Plin C, Tillement JP.
    Biochem Pharmacol; 2004 Nov 15; 68(10):2065-73. PubMed ID: 15476677
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  • 13. The pro-apoptotic proteins, Bid and Bax, cause a limited permeabilization of the mitochondrial outer membrane that is enhanced by cytosol.
    Kluck RM, Esposti MD, Perkins G, Renken C, Kuwana T, Bossy-Wetzel E, Goldberg M, Allen T, Barber MJ, Green DR, Newmeyer DD.
    J Cell Biol; 1999 Nov 15; 147(4):809-22. PubMed ID: 10562282
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  • 14. Beta-phenylethyl isothiocyanate mediated apoptosis; contribution of Bax and the mitochondrial death pathway.
    Rose P, Armstrong JS, Chua YL, Ong CN, Whiteman M.
    Int J Biochem Cell Biol; 2005 Jan 15; 37(1):100-19. PubMed ID: 15381154
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  • 16. Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC.
    Shimizu S, Narita M, Tsujimoto Y.
    Nature; 1999 Jun 03; 399(6735):483-7. PubMed ID: 10365962
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  • 17. Bcl-2 prolongs cell survival after Bax-induced release of cytochrome c.
    Rossé T, Olivier R, Monney L, Rager M, Conus S, Fellay I, Jansen B, Borner C.
    Nature; 1998 Jan 29; 391(6666):496-9. PubMed ID: 9461218
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  • 18. BNIP3 and genetic control of necrosis-like cell death through the mitochondrial permeability transition pore.
    Vande Velde C, Cizeau J, Dubik D, Alimonti J, Brown T, Israels S, Hakem R, Greenberg AH.
    Mol Cell Biol; 2000 Aug 29; 20(15):5454-68. PubMed ID: 10891486
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  • 20. Mitochondrial cytochrome c release is caspase-dependent and does not involve mitochondrial permeability transition in didemnin B-induced apoptosis.
    Grubb DR, Ly JD, Vaillant F, Johnson KL, Lawen A.
    Oncogene; 2001 Jul 05; 20(30):4085-94. PubMed ID: 11494136
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