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Journal Abstract Search

117 related items for PubMed ID: 9765899

  • 21. Heterozygous SOD1 D90A mutation presenting as slowly progressive predominant upper motor neuron amyotrophic lateral sclerosis.
    Luigetti M, Conte A, Madia F, Marangi G, Zollino M, Mancuso I, Dileone M, Del Grande A, Di Lazzaro V, Tonali PA, Sabatelli M.
    Neurol Sci; 2009 Dec; 30(6):517-20. PubMed ID: 19685200
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  • 22. Toxicity of ALS-linked SOD1 mutants.
    Williamson TL, Corson LB, Huang L, Burlingame A, Liu J, Bruijn LI, Cleveland DW.
    Science; 2000 Apr 21; 288(5465):399. PubMed ID: 10798964
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  • 24. A clinicopathological study of patient with familial amyotrophic lateral sclerosis associated with a two-base pair deletion in the copper/zinc superoxide dismutase (SOD1) gene.
    Kadekawa J, Fujimura H, Yanagihara T, Sakoda S.
    Acta Neuropathol; 2001 Apr 21; 101(4):415. PubMed ID: 11355314
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  • 26. Abnormality of Cu/Zn superoxide dismutase (SOD1) activity in Japanese familial amyotrophic lateral sclerosis with two base pair deletion in the SOD1 gene.
    Nakashima K, Watanabe Y, Kuno N, Nanba E, Takahashi K.
    Neurology; 1995 May 21; 45(5):1019-20. PubMed ID: 7746377
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  • 30. Many roads lead to Rome? Multiple modes of Cu,Zn superoxide dismutase destabilization, misfolding and aggregation in amyotrophic lateral sclerosis.
    Broom HR, Rumfeldt JA, Meiering EM.
    Essays Biochem; 2014 May 21; 56():149-65. PubMed ID: 25131593
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  • 32. Chaperone-facilitated copper binding is a property common to several classes of familial amyotrophic lateral sclerosis-linked superoxide dismutase mutants.
    Corson LB, Strain JJ, Culotta VC, Cleveland DW.
    Proc Natl Acad Sci U S A; 1998 May 26; 95(11):6361-6. PubMed ID: 9600970
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  • 33. Neuronal tissue-specific ribonucleoprotein complex formation on SOD1 mRNA: alterations by ALS SOD1 mutations.
    Ge WW, Leystra-Lantz C, Sanelli TR, McLean J, Wen W, Strong W, Strong MJ.
    Neurobiol Dis; 2006 Aug 26; 23(2):342-50. PubMed ID: 16730180
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  • 34. Overexpression of Abeta is associated with acceleration of onset of motor impairment and superoxide dismutase 1 aggregation in an amyotrophic lateral sclerosis mouse model.
    Li QX, Mok SS, Laughton KM, McLean CA, Volitakis I, Cherny RA, Cheung NS, White AR, Masters CL.
    Aging Cell; 2006 Apr 26; 5(2):153-65. PubMed ID: 16626394
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  • 35. Cu/Zn superoxide dismutase (SOD1) mutations associated with familial amyotrophic lateral sclerosis (ALS) affect cellular free radical release in the presence of oxidative stress.
    Cookson MR, Menzies FM, Manning P, Eggett CJ, Figlewicz DA, McNeil CJ, Shaw PJ.
    Amyotroph Lateral Scler Other Motor Neuron Disord; 2002 Jun 26; 3(2):75-85. PubMed ID: 12215229
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  • 36. Mutations in the copper- and zinc-containing superoxide dismutase gene are associated with "Lou Gehrig's disease".
    Nutr Rev; 1993 Aug 26; 51(8):243-5. PubMed ID: 8302496
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  • 37. Caspase-1 is activated in neural cells and tissue with amyotrophic lateral sclerosis-associated mutations in copper-zinc superoxide dismutase.
    Pasinelli P, Borchelt DR, Houseweart MK, Cleveland DW, Brown RH.
    Proc Natl Acad Sci U S A; 1998 Dec 22; 95(26):15763-8. PubMed ID: 9861044
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  • 38. Inclusions of amyotrophic lateral sclerosis-linked superoxide dismutase in ventral horns, liver, and kidney.
    Jonsson PA, Bergemalm D, Andersen PM, Gredal O, Brännström T, Marklund SL.
    Ann Neurol; 2008 May 22; 63(5):671-5. PubMed ID: 18409196
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  • 39. Identification of three mutations in the Cu,Zn-superoxide dismutase (Cu,Zn-SOD) gene with familial amyotrophic lateral sclerosis: transduction of human Cu,Zn-SOD into PC12 cells by HIV-1 TAT protein basic domain.
    Chou CM, Huang CJ, Shih CM, Chen YP, Liu TP, Chen CT.
    Ann N Y Acad Sci; 2005 May 22; 1042():303-13. PubMed ID: 15965076
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  • 40. Increased 3-nitrotyrosine and oxidative damage in mice with a human copper/zinc superoxide dismutase mutation.
    Ferrante RJ, Shinobu LA, Schulz JB, Matthews RT, Thomas CE, Kowall NW, Gurney ME, Beal MF.
    Ann Neurol; 1997 Sep 22; 42(3):326-34. PubMed ID: 9307254
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