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Journal Abstract Search

95 related items for PubMed ID: 9788659

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  • 2. IL-1beta and IL-6 act synergistically with TNF-alpha to alter cardiac contractile function after burn trauma.
    Maass DL, White J, Horton JW.
    Shock; 2002 Oct; 18(4):360-6. PubMed ID: 12392281
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  • 7. Role of cytosolic vs. mitochondrial Ca2+ accumulation in burn injury-related myocardial inflammation and function.
    Maass DL, White J, Sanders B, Horton JW.
    Am J Physiol Heart Circ Physiol; 2005 Feb; 288(2):H744-51. PubMed ID: 15388497
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  • 11. Subchronic exposure of cardiomyocytes to low concentrations of tumor necrosis factor alpha attenuates the positive inotropic response not only to catecholamines but also to cardiac glycosides and high calcium concentrations.
    Boekstegers P, Kainz I, Giehrl W, Peter W, Werdan K.
    Mol Cell Biochem; 1996 Mar 23; 156(2):135-43. PubMed ID: 9095470
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  • 12. Rho-associated kinase modulates myocardial inflammatory cytokine responses.
    Horton JW, Maass DL, Ballard-Croft C.
    Shock; 2005 Jul 23; 24(1):53-8. PubMed ID: 15988321
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  • 15. Hypertonic saline-dextran suppresses burn-related cytokine secretion by cardiomyocytes.
    Horton JW, Maass DL, White J, Sanders B.
    Am J Physiol Heart Circ Physiol; 2001 Apr 23; 280(4):H1591-601. PubMed ID: 11247769
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  • 20. Propofol and ketamine only inhibit intracellular Ca2+ transients and contraction in rat ventricular myocytes at supraclinical concentrations.
    Kanaya N, Murray PA, Damron DS.
    Anesthesiology; 1998 Mar 23; 88(3):781-91. PubMed ID: 9523824
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