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139 related items for PubMed ID: 9789805

  • 1. Acetylcholinesterase: C-terminal domains, molecular forms and functional localization.
    Massoulié J, Anselmet A, Bon S, Krejci E, Legay C, Morel N, Simon S.
    J Physiol Paris; 1998; 92(3-4):183-90. PubMed ID: 9789805
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  • 2. The origin of the molecular diversity and functional anchoring of cholinesterases.
    Massoulié J.
    Neurosignals; 2002; 11(3):130-43. PubMed ID: 12138250
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  • 10. Human endplate acetylcholinesterase deficiency caused by mutations in the collagen-like tail subunit (ColQ) of the asymmetric enzyme.
    Ohno K, Brengman J, Tsujino A, Engel AG.
    Proc Natl Acad Sci U S A; 1998 Aug 04; 95(16):9654-9. PubMed ID: 9689136
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  • 11. Trimerization domain of the collagen tail of acetylcholinesterase.
    Bon S, Ayon A, Leroy J, Massoulié J.
    Neurochem Res; 2003 Apr 04; 28(3-4):523-35. PubMed ID: 12675141
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  • 16. The C-terminal t peptide of acetylcholinesterase forms an alpha helix that supports homomeric and heteromeric interactions.
    Bon S, Dufourcq J, Leroy J, Cornut I, Massoulié J.
    Eur J Biochem; 2004 Jan 04; 271(1):33-47. PubMed ID: 14686917
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  • 17. The synaptic acetylcholinesterase tetramer assembles around a polyproline II helix.
    Dvir H, Harel M, Bon S, Liu WQ, Vidal M, Garbay C, Sussman JL, Massoulié J, Silman I.
    EMBO J; 2004 Nov 10; 23(22):4394-405. PubMed ID: 15526038
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  • 18. Acetylcholinesterase H and T dimers are associated through the same contact. Mutations at this interface interfere with the C-terminal T peptide, inducing degradation rather than secretion.
    Morel N, Leroy J, Ayon A, Massoulié J, Bon S.
    J Biol Chem; 2001 Oct 05; 276(40):37379-89. PubMed ID: 11443120
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  • 19. Elements of the C-terminal t peptide of acetylcholinesterase that determine amphiphilicity, homomeric and heteromeric associations, secretion and degradation.
    Belbeoc'h S, Falasca C, Leroy J, Ayon A, Massoulié J, Bon S.
    Eur J Biochem; 2004 Apr 05; 271(8):1476-87. PubMed ID: 15066173
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  • 20. Remodeling of the neuromuscular junction in mice with deleted exons 5 and 6 of acetylcholinesterase.
    Girard E, Bernard V, Camp S, Taylor P, Krejci E, Molgó J.
    J Mol Neurosci; 2006 Apr 05; 30(1-2):99-100. PubMed ID: 17192646
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