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Journal Abstract Search

203 related items for PubMed ID: 9794433

  • 1. Nicotinamide-adenine dinucleotide phosphate oxidase assembly and activation in EBV-transformed B lymphoblastoid cell lines of normal and chronic granulomatous disease patients.
    Dusi S, Nadalini KA, Donini M, Zentilin L, Wientjes FB, Roos D, Giacca M, Rossi F.
    J Immunol; 1998 Nov 01; 161(9):4968-74. PubMed ID: 9794433
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  • 4. NADPH oxidase activity and cytochrome b558 content of human Epstein-Barr-virus-transformed B lymphocytes correlate with expression of genes encoding components of the oxidase system.
    Condino-Neto A, Newburger PE.
    Arch Biochem Biophys; 1998 Dec 15; 360(2):158-64. PubMed ID: 9851826
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  • 5. Mechanisms of NADPH oxidase activation: translocation of p40phox, Rac1 and Rac2 from the cytosol to the membranes in human neutrophils lacking p47phox or p67phox.
    Dusi S, Donini M, Rossi F.
    Biochem J; 1996 Mar 01; 314 ( Pt 2)(Pt 2):409-12. PubMed ID: 8670049
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  • 7. Rac translocates independently of the neutrophil NADPH oxidase components p47phox and p67phox. Evidence for its interaction with flavocytochrome b558.
    Heyworth PG, Bohl BP, Bokoch GM, Curnutte JT.
    J Biol Chem; 1994 Dec 09; 269(49):30749-52. PubMed ID: 7982999
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  • 8. P40phox associates with the neutrophil Triton X-100-insoluble cytoskeletal fraction and PMA-activated membrane skeleton: a comparative study with P67phox and P47phox.
    El Benna J, Dang PM, Andrieu V, Vergnaud S, Dewas C, Cachia O, Fay M, Morel F, Chollet-Martin S, Hakim J, Gougerot-Pocidalo MA.
    J Leukoc Biol; 1999 Dec 09; 66(6):1014-20. PubMed ID: 10614785
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  • 9. Restitution of superoxide generation in autosomal cytochrome-negative chronic granulomatous disease (A22(0) CGD)-derived B lymphocyte cell lines by transfection with p22phax cDNA.
    Maly FE, Schuerer-Maly CC, Quilliam L, Cochrane CG, Newburger PE, Curnutte JT, Gifford M, Dinauer MC.
    J Exp Med; 1993 Dec 01; 178(6):2047-53. PubMed ID: 8245781
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  • 10. The superoxide-generating NADPH oxidase: structural aspects and activation mechanism.
    Vignais PV.
    Cell Mol Life Sci; 2002 Sep 01; 59(9):1428-59. PubMed ID: 12440767
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  • 11. The molecular basis of chronic granulomatous disease.
    Meischl C, Roos D.
    Springer Semin Immunopathol; 1998 Sep 01; 19(4):417-34. PubMed ID: 9618766
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  • 13. NADPH-binding component of the respiratory burst oxidase system: studies using neutrophil membranes from patients with chronic granulomatous disease lacking the beta-subunit of cytochrome b558.
    Tsunawaki S, Mizunari H, Namiki H, Kuratsuji T.
    J Exp Med; 1994 Jan 01; 179(1):291-7. PubMed ID: 8270871
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  • 14. Intersecting Stories of the Phagocyte NADPH Oxidase and Chronic Granulomatous Disease.
    Nauseef WM, Clark RA.
    Methods Mol Biol; 2019 Jan 01; 1982():3-16. PubMed ID: 31172463
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  • 17. X-linked chronic granulomatous disease: correction of NADPH oxidase defect by retrovirus-mediated expression of gp91-phox.
    Porter CD, Parkar MH, Levinsky RJ, Collins MK, Kinnon C.
    Blood; 1993 Oct 01; 82(7):2196-202. PubMed ID: 8400270
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  • 18. Mechanisms of NADPH oxidase activation in human neutrophils: p67phox is required for the translocation of rac 1 but not of rac 2 from cytosol to the membranes.
    Dusi S, Donini M, Rossi F.
    Biochem J; 1995 Jun 15; 308 ( Pt 3)(Pt 3):991-4. PubMed ID: 8948460
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  • 19. Cytosolic guanine nucleotide-binding protein Rac2 operates in vivo as a component of the neutrophil respiratory burst oxidase. Transfer of Rac2 and the cytosolic oxidase components p47phox and p67phox to the submembranous actin cytoskeleton during oxidase activation.
    el Benna J, Ruedi JM, Babior BM.
    J Biol Chem; 1994 Mar 04; 269(9):6729-34. PubMed ID: 8120032
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