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2. Glutaminergic vagal afferents may mediate both retching and gastric adaptive relaxation in dogs. Furukawa N, Hatano M, Fukuda H. Auton Neurosci; 2001 Oct 08; 93(1-2):21-30. PubMed ID: 11695702 [Abstract] [Full Text] [Related]
6. Non-N-methyl-D-aspartate receptors may mediate the transmission of emetic signals between visceral vagal afferents and the solitary nucleus in dogs. Furukawa N, Hatano M, Fukuda H, Koga T. Neurosci Lett; 1998 Dec 11; 258(1):53-6. PubMed ID: 9876050 [Abstract] [Full Text] [Related]
10. The site of the anti-emetic action of tachykinin NK1 receptor antagonists may exist in the medullary area adjacent to the semicompact part of the nucleus ambiguus. Fukuda H, Nakamura E, Koga T, Furukawa N, Shiroshita Y. Brain Res; 1999 Feb 13; 818(2):439-49. PubMed ID: 10082830 [Abstract] [Full Text] [Related]
19. Hypercapnia and hypoxia which develop during retching participate in the transition from retching to expulsion in dogs. Fukuda H, Koga T. Neurosci Res; 1993 Aug 13; 17(3):205-15. PubMed ID: 8233124 [Abstract] [Full Text] [Related]
20. The nucleus raphe magnus suppresses vomiting, and the solitary nucleus and 5-HT are not involved in this suppression. Hattori Y, Hamaguchi C, Yamada Y, Urayama Y, Nakamura E, Koga T, Fukuda H. Auton Neurosci; 2010 Jan 15; 152(1-2):41-8. PubMed ID: 19773202 [Abstract] [Full Text] [Related] Page: [Next] [New Search]