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137 related items for PubMed ID: 9822260
1. Anti-TNF treatment of baboons with sepsis reduces TNF-alpha, IL-6 and IL-8, but not the degree of complement activation. Bengtsson A, Redl H, Schlag G, Högåsen K, Götze O, Mollnes TE. Scand J Immunol; 1998 Nov; 48(5):509-14. PubMed ID: 9822260 [Abstract] [Full Text] [Related]
5. Inhibition of factor XII in septic baboons attenuates the activation of complement and fibrinolytic systems and reduces the release of interleukin-6 and neutrophil elastase. Jansen PM, Pixley RA, Brouwer M, de Jong IW, Chang AC, Hack CE, Taylor FB, Colman RW. Blood; 1996 Mar 15; 87(6):2337-44. PubMed ID: 8630396 [Abstract] [Full Text] [Related]
9. Complement and leukocyte activation in septic baboons. Bengtsson A, Redl H, Paul E, Schlag G, Mollnes TE, Davies J. Circ Shock; 1993 Feb 15; 39(2):83-8. PubMed ID: 8490996 [Abstract] [Full Text] [Related]
10. Endogenous modulators of TNF and IL-1 response are under partial control of TNF in baboon bacteremia. Redl H, Schlag G, Paul E, Bahrami S, Buurman WA, Strieter RM, Kunkel SL, Davies J, Foulkes R. Am J Physiol; 1996 Nov 15; 271(5 Pt 2):R1193-8. PubMed ID: 8945953 [Abstract] [Full Text] [Related]
13. Anti-L-selectin antibody therapy does not worsen the postseptic course in a baboon model. Redl HR, Martin U, Khadem A, Pelinka LE, van Griensven M. Crit Care; 2005 Nov 15; 9(6):R735-44. PubMed ID: 16356222 [Abstract] [Full Text] [Related]