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Journal Abstract Search

194 related items for PubMed ID: 9849960

  • 1. Certain activating mutations within helix 6 of the human luteinizing hormone receptor may be explained by alterations that allow transmembrane regions to activate Gs.
    Abell AN, McCormick DJ, Segaloff DL.
    Mol Endocrinol; 1998 Dec; 12(12):1857-69. PubMed ID: 9849960
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  • 4. Genetic heterogeneity of constitutively activating mutations of the human luteinizing hormone receptor in familial male-limited precocious puberty.
    Laue L, Chan WY, Hsueh AJ, Kudo M, Hsu SY, Wu SM, Blomberg L, Cutler GB.
    Proc Natl Acad Sci U S A; 1995 Mar 14; 92(6):1906-10. PubMed ID: 7892197
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  • 5. Phe576 plays an important role in the secondary structure and intracellular signaling of the human luteinizing hormone/chorionic gonadotropin receptor.
    Yano K, Kohn LD, Saji M, Okuno A, Cutler GB.
    J Clin Endocrinol Metab; 1997 Aug 14; 82(8):2586-91. PubMed ID: 9253338
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  • 6. Pleiotropic effects of substitutions of a highly conserved leucine in transmembrane helix III of the human lutropin/choriogonadotropin receptor with respect to constitutive activation and hormone responsiveness.
    Shinozaki H, Fanelli F, Liu X, Jaquette J, Nakamura K, Segaloff DL.
    Mol Endocrinol; 2001 Jun 14; 15(6):972-84. PubMed ID: 11376115
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  • 7. Evidence for the direct involvement of transmembrane region 6 of the lutropin/choriogonadotropin receptor in activating Gs.
    Abell AN, Segaloff DL.
    J Biol Chem; 1997 Jun 06; 272(23):14586-91. PubMed ID: 9169417
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  • 8. A novel luteinizing hormone receptor mutation in a patient with familial male-limited precocious puberty: effect of the size of a critical amino acid on receptor activity.
    Wu SM, Leschek EW, Brain C, Chan WY.
    Mol Genet Metab; 1999 Jan 06; 66(1):68-73. PubMed ID: 9973550
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  • 10. [The secondary structure of peptides derived from the third intracellular loop of the serpentine type receptors and its interrelation with their biological activity].
    Shpakova EA, Skvortsova EA, Tarasenko II, Shpakov AO.
    Tsitologiia; 2012 Jan 06; 54(2):119-29. PubMed ID: 22590924
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  • 12. A new constitutively activating point mutation in the luteinizing hormone/choriogonadotropin receptor gene in cases of male-limited precocious puberty.
    Yano K, Saji M, Hidaka A, Moriya N, Okuno A, Kohn LD, Cutler GB.
    J Clin Endocrinol Metab; 1995 Apr 06; 80(4):1162-8. PubMed ID: 7714085
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  • 14. The regulation of the binding affinity of the luteinizing hormone/choriogonadotropin receptor by sodium ions is mediated by a highly conserved aspartate located in the second transmembrane domain of G protein-coupled receptors.
    Quintana J, Wang H, Ascoli M.
    Mol Endocrinol; 1993 Jun 06; 7(6):767-75. PubMed ID: 8395653
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  • 16. The formation of a salt bridge between helices 3 and 6 is responsible for the constitutive activity and lack of hormone responsiveness of the naturally occurring L457R mutation of the human lutropin receptor.
    Zhang M, Mizrachi D, Fanelli F, Segaloff DL.
    J Biol Chem; 2005 Jul 15; 280(28):26169-76. PubMed ID: 15908694
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  • 19. Mutagenesis within helix 6 of the human beta1-adrenergic receptor identifies Lysine324 as a residue involved in imparting the high-affinity binding state of agonists.
    Zeitoun O, Santos NM, Gardner LA, White SW, Bahouth SW.
    Mol Pharmacol; 2006 Sep 15; 70(3):838-50. PubMed ID: 16760361
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