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Journal Abstract Search

251 related items for PubMed ID: 9852064

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  • 2. Long QT syndrome-associated mutations in the S4-S5 linker of KvLQT1 potassium channels modify gating and interaction with minK subunits.
    Franqueza L, Lin M, Shen J, Splawski I, Keating MT, Sanguinetti MC.
    J Biol Chem; 1999 Jul 23; 274(30):21063-70. PubMed ID: 10409658
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  • 4. Single-channel characteristics of wild-type IKs channels and channels formed with two minK mutants that cause long QT syndrome.
    Sesti F, Goldstein SA.
    J Gen Physiol; 1998 Dec 23; 112(6):651-63. PubMed ID: 9834138
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  • 5. Activation and inactivation of homomeric KvLQT1 potassium channels.
    Pusch M, Magrassi R, Wollnik B, Conti F.
    Biophys J; 1998 Aug 23; 75(2):785-92. PubMed ID: 9675180
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  • 7. Molecular mechanism and functional significance of the MinK control of the KvLQT1 channel activity.
    Romey G, Attali B, Chouabe C, Abitbol I, Guillemare E, Barhanin J, Lazdunski M.
    J Biol Chem; 1997 Jul 04; 272(27):16713-6. PubMed ID: 9201970
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  • 10. A recessive C-terminal Jervell and Lange-Nielsen mutation of the KCNQ1 channel impairs subunit assembly.
    Schmitt N, Schwarz M, Peretz A, Abitbol I, Attali B, Pongs O.
    EMBO J; 2000 Feb 01; 19(3):332-40. PubMed ID: 10654932
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  • 12. Inhibition of cardiac delayed rectifier K+ currents by an antisense oligodeoxynucleotide against IsK (minK) and over-expression of IsK mutant D77N in neonatal mouse hearts.
    Ohyama H, Kajita H, Omori K, Takumi T, Hiramoto N, Iwasaka T, Matsuda H.
    Pflugers Arch; 2001 Jun 01; 442(3):329-35. PubMed ID: 11484762
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  • 14. Stereoselective interactions of the enantiomers of chromanol 293B with human voltage-gated potassium channels.
    Yang IC, Scherz MW, Bahinski A, Bennett PB, Murray KT.
    J Pharmacol Exp Ther; 2000 Sep 01; 294(3):955-62. PubMed ID: 10945846
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  • 15. Voltage-dependent inactivation of the human K+ channel KvLQT1 is eliminated by association with minimal K+ channel (minK) subunits.
    Tristani-Firouzi M, Sanguinetti MC.
    J Physiol; 1998 Jul 01; 510 ( Pt 1)(Pt 1):37-45. PubMed ID: 9625865
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  • 16. Single-channel properties of IKs potassium channels.
    Yang Y, Sigworth FJ.
    J Gen Physiol; 1998 Dec 01; 112(6):665-78. PubMed ID: 9834139
    [Abstract] [Full Text] [Related]

  • 17. Dominant-negative KvLQT1 mutations underlie the LQT1 form of long QT syndrome.
    Shalaby FY, Levesque PC, Yang WP, Little WA, Conder ML, Jenkins-West T, Blanar MA.
    Circulation; 1997 Sep 16; 96(6):1733-6. PubMed ID: 9323054
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  • 18. Pathophysiological mechanisms of dominant and recessive KVLQT1 K+ channel mutations found in inherited cardiac arrhythmias.
    Wollnik B, Schroeder BC, Kubisch C, Esperer HD, Wieacker P, Jentsch TJ.
    Hum Mol Genet; 1997 Oct 16; 6(11):1943-9. PubMed ID: 9302275
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  • 19. A dominant negative isoform of the long QT syndrome 1 gene product.
    Demolombe S, Baró I, Péréon Y, Bliek J, Mohammad-Panah R, Pollard H, Morid S, Mannens M, Wilde A, Barhanin J, Charpentier F, Escande D.
    J Biol Chem; 1998 Mar 20; 273(12):6837-43. PubMed ID: 9506986
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  • 20. Cocaine blocks HERG, but not KvLQT1+minK, potassium channels.
    Zhang S, Rajamani S, Chen Y, Gong Q, Rong Y, Zhou Z, Ruoho A, January CT.
    Mol Pharmacol; 2001 May 20; 59(5):1069-76. PubMed ID: 11306689
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