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Journal Abstract Search
199 related items for PubMed ID: 9864159
1. Mild hemophilia A caused by increased rate of factor VIII A2 subunit dissociation: evidence for nonproteolytic inactivation of factor VIIIa in vivo. Pipe SW, Eickhorst AN, McKinley SH, Saenko EL, Kaufman RJ. Blood; 1999 Jan 01; 93(1):176-83. PubMed ID: 9864159 [Abstract] [Full Text] [Related]
2. Hemophilia A mutations associated with 1-stage/2-stage activity discrepancy disrupt protein-protein interactions within the triplicated A domains of thrombin-activated factor VIIIa. Pipe SW, Saenko EL, Eickhorst AN, Kemball-Cook G, Kaufman RJ. Blood; 2001 Feb 01; 97(3):685-91. PubMed ID: 11157485 [Abstract] [Full Text] [Related]
11. Mutations associated with hemophilia A in the 558-565 loop of the factor VIIIa A2 subunit alter the catalytic activity of the factor Xase complex. Jenkins PV, Freas J, Schmidt KM, Zhou Q, Fay PJ. Blood; 2002 Jul 15; 100(2):501-8. PubMed ID: 12091341 [Abstract] [Full Text] [Related]
13. Contribution of A1 subunit residue Q316 in thrombin-activated factor VIII to A2 subunit dissociation. Parker ET, Lollar P. Biochemistry; 2007 Aug 28; 46(34):9737-42. PubMed ID: 17676877 [Abstract] [Full Text] [Related]
16. Acidic Region Residues 1680-1684 in the A3 Domain of Factor VIII Contain a Thrombin-Interactive Site Responsible for Proteolytic Cleavage at Arg1689. Nakajima Y, Minami H, Nogami K. Thromb Haemost; 2021 Oct 28; 121(10):1274-1288. PubMed ID: 33592631 [Abstract] [Full Text] [Related]