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Journal Abstract Search
177 related items for PubMed ID: 9914780
1. Inhibition of early-phase exogenous and endogenous liver carcinogenesis in transgenic rats harboring a rat glutathione S-transferase placental form gene. Nakae D, Denda A, Kobayashi Y, Akai H, Kishida H, Tsujiuchi T, Konishi Y, Suzuki T, Muramatsu M. Jpn J Cancer Res; 1998 Nov; 89(11):1118-25. PubMed ID: 9914780 [Abstract] [Full Text] [Related]
4. Prevention by 1'-acetoxychavicol acetate of the induction but not growth of putative preneoplastic, glutathione S-transferase placental form-positive, focal lesions in the livers of rats fed a choline-deficient, L-amino acid-defined diet. Kobayashi Y, Nakae D, Akai H, Kishida H, Okajima E, Kitayama W, Denda A, Tsujiuchi T, Murakami A, Koshimizu K, Ohigashi H, Konishi Y. Carcinogenesis; 1998 Oct; 19(10):1809-14. PubMed ID: 9806163 [Abstract] [Full Text] [Related]
6. Biphasic modifying effect of indole-3-carbinol on diethylnitrosamine-induced preneoplastic glutathione S-transferase placental form-positive liver cell foci in Sprague-Dawley rats. Kim DJ, Lee KK, Han BS, Ahn B, Bae JH, Jang JJ. Jpn J Cancer Res; 1994 Jun; 85(6):578-83. PubMed ID: 8063610 [Abstract] [Full Text] [Related]
7. Stable phenotypic expression of glutathione S-transferase placental type and unstable phenotypic expression of gamma-glutamyltransferase in rat liver preneoplastic and neoplastic lesions. Tatematsu M, Mera Y, Inoue T, Satoh K, Sato K, Ito N. Carcinogenesis; 1988 Feb; 9(2):215-20. PubMed ID: 2892592 [Abstract] [Full Text] [Related]
9. Transgenic disruption of gap junctional intercellular communication enhances early but not late stage hepatocarcinogenesis in the rat. Hokaiwado N, Asamoto M, Ogawa K, Shirai T. Toxicol Pathol; 2005 Feb; 33(6):695-701. PubMed ID: 16243774 [Abstract] [Full Text] [Related]
10. Correlation between S-adenosyl-L-methionine content and production of c-myc, c-Ha-ras, and c-Ki-ras mRNA transcripts in the early stages of rat liver carcinogenesis. Simile MM, Pascale R, De Miglio MR, Nufris A, Daino L, Seddaiu MA, Gaspa L, Feo F. Cancer Lett; 1994 Apr 29; 79(1):9-16. PubMed ID: 7910516 [Abstract] [Full Text] [Related]
11. Combined supplementation of vanadium and beta-carotene suppresses placental glutathione S-transferase-positive foci and enhances antioxidant functions during the inhibition of diethylnitrosamine-induced rat liver carcinogenesis. Chattopadhyay MB, C B MK, Kanna PS, Ray RS, Roy S, Chatterjee M. J Gastroenterol Hepatol; 2004 Jun 29; 19(6):683-93. PubMed ID: 15151625 [Abstract] [Full Text] [Related]
12. Role of diethylnitrosamine, 2-acetylaminofluorene and partial hepatectomy in the expression of glutathione-S-transferase-P and gamma-glutamyltranspeptidase in the early steps of rat liver carcinogenesis. Ould Elhkim M, Decloître F, Martin M, Loridon-Rosa B, Frayssinet C. Tumour Biol; 1992 Jun 29; 13(3):152-61. PubMed ID: 1352647 [Abstract] [Full Text] [Related]
13. Effect of nodularin on the expression of glutathione S-transferase placental form and proliferating cell nuclear antigen in N-nitrosodiethylamine initiated hepatocarcinogenesis in the male Fischer 344 rat. Song KY, Lim IK, Park SC, Lee SO, Park HS, Choi YK, Hyun BH. Carcinogenesis; 1999 Aug 29; 20(8):1541-8. PubMed ID: 10426804 [Abstract] [Full Text] [Related]
14. Decreased expression of Bcl-x protein during hepatocarcinogenesis induced exogenously and endogenously in rats. Hatanaka Y, Nakae D, Mutai M, Hashizume K, Kamihara Y, Kinoshita N, Tani Y, Danno Gi G, Ohta S, Konishi Y, Ashida H. Jpn J Cancer Res; 2001 Dec 29; 92(12):1270-7. PubMed ID: 11749691 [Abstract] [Full Text] [Related]
15. Immunohistochemical demonstration of the gap junctional protein connexin 32 and proliferating cell nuclear antigen in glutathione S-transferase placental form-negative lesions of rat liver induced by diethylnitrosamine and clofibrate. Ito S, Tateno C, Tuda M, Yoshitake A. Toxicol Pathol; 1996 Dec 29; 24(6):690-5. PubMed ID: 8994295 [Abstract] [Full Text] [Related]
16. Inhibition by acetylsalicylic acid, a cyclo-oxygenase inhibitor, and p-bromophenacylbromide, a phospholipase A2 inhibitor, of both cirrhosis and enzyme-altered nodules caused by a choline-deficient, L-amino acid-defined diet in rats. Endoh T, Tang Q, Denda A, Noguchi O, Kobayashi E, Tamura K, Horiguchi K, Ogasawara H, Tsujiuchi T, Nakae D, Sugimura M, Konishi Y. Carcinogenesis; 1996 Mar 29; 17(3):467-75. PubMed ID: 8631132 [Abstract] [Full Text] [Related]
18. Inhibitory effect of α-lipoic acid on thioacetamide-induced tumor promotion through suppression of inflammatory cell responses in a two-stage hepatocarcinogenesis model in rats. Fujii Y, Segawa R, Kimura M, Wang L, Ishii Y, Yamamoto R, Morita R, Mitsumori K, Shibutani M. Chem Biol Interact; 2013 Sep 25; 205(2):108-18. PubMed ID: 23830814 [Abstract] [Full Text] [Related]
19. Reduction of glutathione S-transferase P-form mRNA expression in remodeling nodules in rat liver revealed by in situ hybridization. Imai T, Masui T, Ichinose M, Nakanishi H, Yanai T, Masegi T, Muramatsu M, Tatematsu M. Carcinogenesis; 1997 Mar 25; 18(3):545-51. PubMed ID: 9067555 [Abstract] [Full Text] [Related]