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Journal Abstract Search

234 related items for PubMed ID: 9916703

  • 1. Persistence of autoreactive T cell drive is required to elicit anti-chromatin antibodies in a murine model of drug-induced lupus.
    Kretz-Rommel A, Rubin RL.
    J Immunol; 1999 Jan 15; 162(2):813-20. PubMed ID: 9916703
    [Abstract] [Full Text] [Related]

  • 2. Autoimmunity caused by disruption of central T cell tolerance. A murine model of drug-induced lupus.
    Kretz-Rommel A, Duncan SR, Rubin RL.
    J Clin Invest; 1997 Apr 15; 99(8):1888-96. PubMed ID: 9109433
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  • 3. Early cellular events in systemic autoimmunity driven by chromatin-reactive T cells.
    Kretz-Rommel A, Rubin RL.
    Cell Immunol; 2001 Mar 15; 208(2):125-36. PubMed ID: 11333145
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  • 4. A nondeletional mechanism for central T-cell tolerance.
    Rubin RL, Kretz-Rommel A.
    Crit Rev Immunol; 2001 Mar 15; 21(1-3):29-40. PubMed ID: 11642611
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  • 5. CD4+ T cell lines with selective patterns of autoreactivity as well as CD4- CD8- T helper cell lines augment the production of idiotypes shared by pathogenic anti-DNA autoantibodies in the NZB x SWR model of lupus nephritis.
    Sainis K, Datta SK.
    J Immunol; 1988 Apr 01; 140(7):2215-24. PubMed ID: 2965184
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  • 10. Autoimmunization in murine graft-vs-host disease. I. Selective production of antibodies to histones and DNA.
    Portanova JP, Claman HN, Kotzin BL.
    J Immunol; 1985 Dec 01; 135(6):3850-6. PubMed ID: 3877758
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  • 12. Autoimmune syndrome after induction of neonatal tolerance to alloantigens: effects of in vivo treatment with anti-T cell subset monoclonal antibodies.
    Merino J, Schurmans S, Luzuy S, Izui S, Vassalli P, Lambert PH.
    J Immunol; 1987 Sep 01; 139(5):1426-31. PubMed ID: 3114366
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  • 13. Attempts at standardization of lupus-like graft-vs-host disease: inadvertent repopulation by DBA/2 spleen cells of H-2-different nonirradiated F1 mice.
    Van Rappard-Van Der Veen FM, Radaszkiewicz T, Terraneo L, Gleichmann E.
    J Immunol; 1983 Jun 01; 130(6):2693-701. PubMed ID: 6602176
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  • 14. B7 costimulation in the development of lupus: autoimmunity arises either in the absence of B7.1/B7.2 or in the presence of anti-b7.1/B7.2 blocking antibodies.
    Liang B, Gee RJ, Kashgarian MJ, Sharpe AH, Mamula MJ.
    J Immunol; 1999 Aug 15; 163(4):2322-9. PubMed ID: 10438978
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  • 15. B cell genotype determines the fine specificity of autoantibody in lpr mice.
    Kakkanaiah VN, Sobel ES, MacDonald GC, Cheek RL, Cohen PL, Eisenberg RA.
    J Immunol; 1997 Jul 15; 159(2):1027-35. PubMed ID: 9218626
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  • 16. Transgenic expression of Fas in T cells blocks lymphoproliferation but not autoimmune disease in MRL-lpr mice.
    Fukuyama H, Adachi M, Suematsu S, Miwa K, Suda T, Yoshida N, Nagata S.
    J Immunol; 1998 Apr 15; 160(8):3805-11. PubMed ID: 9558084
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  • 18. Fas and Fas ligand mutations inhibit autoantibody production in pristane-induced lupus.
    Satoh M, Weintraub JP, Yoshida H, Shaheen VM, Richards HB, Shaw M, Reeves WH.
    J Immunol; 2000 Jul 15; 165(2):1036-43. PubMed ID: 10878381
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  • 19. Selective production of autoantibodies in graft-vs-host-induced and spontaneous murine lupus. Predominant reactivity with histone regions accessible in chromatin.
    Portanova JP, Arndt RE, Kotzin BL.
    J Immunol; 1988 Feb 01; 140(3):755-60. PubMed ID: 3339241
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  • 20. Control of spontaneous B lymphocyte autoimmunity with adenovirus-encoded soluble TACI.
    Liu W, Szalai A, Zhao L, Liu D, Martin F, Kimberly RP, Zhou T, Carter RH.
    Arthritis Rheum; 2004 Jun 01; 50(6):1884-96. PubMed ID: 15188365
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