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216 related items for PubMed ID: 9952402

  • 1. Activation of protein kinase A contributes to the expression but not the induction of long-term hyperexcitability caused by axotomy of Aplysia sensory neurons.
    Liao X, Gunstream JD, Lewin MR, Ambron RT, Walters ET.
    J Neurosci; 1999 Feb 15; 19(4):1247-56. PubMed ID: 9952402
    [Abstract] [Full Text] [Related]

  • 2. Evidence that long-term hyperexcitability of the sensory neuron soma induced by nerve injury in Aplysia is adaptive.
    Gasull X, Liao X, Dulin MF, Phelps C, Walters ET.
    J Neurophysiol; 2005 Sep 15; 94(3):2218-30. PubMed ID: 15944238
    [Abstract] [Full Text] [Related]

  • 3. Limited contributions of serotonin to long-term hyperexcitability of Aplysia sensory neurons.
    Liao X, Brou CG, Walters ET.
    J Neurophysiol; 1999 Dec 15; 82(6):3223-35. PubMed ID: 10601456
    [Abstract] [Full Text] [Related]

  • 4. Serotonin induces memory-like, rapamycin-sensitive hyperexcitability in sensory axons of aplysia that contributes to injury responses.
    Weragoda RM, Walters ET.
    J Neurophysiol; 2007 Sep 15; 98(3):1231-9. PubMed ID: 17634332
    [Abstract] [Full Text] [Related]

  • 5. Long-term effects of axotomy on excitability and growth of isolated Aplysia sensory neurons in cell culture: potential role of cAMP.
    Bedi SS, Salim A, Chen S, Glanzman DL.
    J Neurophysiol; 1998 Mar 15; 79(3):1371-83. PubMed ID: 9497418
    [Abstract] [Full Text] [Related]

  • 6. A neuronal isoform of protein kinase G couples mitogen-activated protein kinase nuclear import to axotomy-induced long-term hyperexcitability in Aplysia sensory neurons.
    Sung YJ, Walters ET, Ambron RT.
    J Neurosci; 2004 Aug 25; 24(34):7583-95. PubMed ID: 15329406
    [Abstract] [Full Text] [Related]

  • 7. Activation and retrograde transport of protein kinase G in rat nociceptive neurons after nerve injury and inflammation.
    Sung YJ, Chiu DTW, Ambron RT.
    Neuroscience; 2006 Aug 25; 141(2):697-709. PubMed ID: 16730916
    [Abstract] [Full Text] [Related]

  • 8. Effects of axotomy on cultured sensory neurons of Aplysia: long-term injury-induced changes in excitability and morphology are mediated by different signaling pathways.
    Bedi SS, Cai D, Glanzman DL.
    J Neurophysiol; 2008 Dec 25; 100(6):3209-24. PubMed ID: 18842953
    [Abstract] [Full Text] [Related]

  • 9. Long-term alteration of S-type potassium current and passive membrane properties in aplysia sensory neurons following axotomy.
    Ungless MA, Gasull X, Walters ET.
    J Neurophysiol; 2002 May 25; 87(5):2408-20. PubMed ID: 11976378
    [Abstract] [Full Text] [Related]

  • 10. Memory-like alterations in Aplysia axons after nerve injury or localized depolarization.
    Weragoda RM, Ferrer E, Walters ET.
    J Neurosci; 2004 Nov 17; 24(46):10393-401. PubMed ID: 15548654
    [Abstract] [Full Text] [Related]

  • 11. Retrograde transport of plasticity signals in Aplysia sensory neurons following axonal injury.
    Gunstream JD, Castro GA, Walters ET.
    J Neurosci; 1995 Jan 17; 15(1 Pt 1):439-48. PubMed ID: 7823148
    [Abstract] [Full Text] [Related]

  • 12. Postsynaptic application of a cAMP analogue reverses long-term potentiation in hippocampal CA1 pyramidal neurons.
    Otmakhov N, Lisman JE.
    J Neurophysiol; 2002 Jun 17; 87(6):3018-32. PubMed ID: 12037205
    [Abstract] [Full Text] [Related]

  • 13. Axonal rejoining inhibits injury-induced long-term changes in Aplysia sensory neurons in vitro.
    Bedi SS, Glanzman DL.
    J Neurosci; 2001 Dec 15; 21(24):9667-77. PubMed ID: 11739576
    [Abstract] [Full Text] [Related]

  • 14. Postsynaptic application of a peptide inhibitor of cAMP-dependent protein kinase blocks expression of long-lasting synaptic potentiation in hippocampal neurons.
    Duffy SN, Nguyen PV.
    J Neurosci; 2003 Feb 15; 23(4):1142-50. PubMed ID: 12598602
    [Abstract] [Full Text] [Related]

  • 15. Computational model of the serotonergic modulation of sensory neurons in Aplysia.
    Baxter DA, Canavier CC, Clark JW, Byrne JH.
    J Neurophysiol; 1999 Dec 15; 82(6):2914-35. PubMed ID: 10601429
    [Abstract] [Full Text] [Related]

  • 16. Differential role of mitogen-activated protein kinase in three distinct phases of memory for sensitization in Aplysia.
    Sharma SK, Sherff CM, Shobe J, Bagnall MW, Sutton MA, Carew TJ.
    J Neurosci; 2003 May 01; 23(9):3899-907. PubMed ID: 12736359
    [Abstract] [Full Text] [Related]

  • 17. Effects of activation and inhibition of cAMP-dependent protein kinase on long-term habituation in the crab Chasmagnathus.
    Romano A, Locatelli F, Delorenzi A, Pedreira ME, Maldonado H.
    Brain Res; 1996 Sep 30; 735(1):131-40. PubMed ID: 8905178
    [Abstract] [Full Text] [Related]

  • 18. RISK-1: a novel MAPK homologue in axoplasm that is activated and retrogradely transported after nerve injury.
    Sung YJ, Povelones M, Ambron RT.
    J Neurobiol; 2001 Apr 30; 47(1):67-79. PubMed ID: 11257614
    [Abstract] [Full Text] [Related]

  • 19. Mechanisms for generating the autonomous cAMP-dependent protein kinase required for long-term facilitation in Aplysia.
    Chain DG, Casadio A, Schacher S, Hegde AN, Valbrun M, Yamamoto N, Goldberg AL, Bartsch D, Kandel ER, Schwartz JH.
    Neuron; 1999 Jan 30; 22(1):147-56. PubMed ID: 10027297
    [Abstract] [Full Text] [Related]

  • 20. Roles of CaMKII, PKA, and PKC in the induction and maintenance of LTP of C-fiber-evoked field potentials in rat spinal dorsal horn.
    Yang HW, Hu XD, Zhang HM, Xin WJ, Li MT, Zhang T, Zhou LJ, Liu XG.
    J Neurophysiol; 2004 Mar 30; 91(3):1122-33. PubMed ID: 14586032
    [Abstract] [Full Text] [Related]

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